ReviewJournal of muscle research and cell motility2006
Global and local control of microtubule destabilization promoted by a catastrophe kinesin MCAK/XKCM1.
Review in Journal of muscle research and cell motility, 2006. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 12 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
12 citing papers in PubMed.
- Bioinformatical analysis of the key differentially expressed genes for screening potential biomarkers in Wilms tumor.Scientific reports · 2023Article
- OsKinesin-13A is an active microtubule depolymerase involved in glume length regulation via affecting cell elongation.Scientific reports · 2015Article
- Stimulation of the CLIP-170--dependent capture of membrane organelles by microtubules through fine tuning of microtubule assembly dynamics.Molecular biology of the cell · 2011Article
- Analysis of Dictyostelium TACC reveals differential interactions with CP224 and unusual dynamics of Dictyostelium microtubules.Cellular and molecular life sciences : CMLS · 2011Article
- Mitotic regulation of the stability of microtubule plus-end tracking protein EB3 by ubiquitin ligase SIAH-1 and Aurora mitotic kinases.The Journal of biological chemistry · 2009Article
- Predominant regulators of tubulin monomer-polymer partitioning and their implication for cell polarization.Cellular and molecular life sciences : CMLS · 2009Review
- Role of APC and its binding partners in regulating microtubules in mitosis.Advances in experimental medicine and biology · 2009Review
- Age-dependent changes in neuronal distribution of CacyBP/SIP: comparison to tubulin and the tau protein.Journal of neural transmission (Vienna, Austria : 1996) · 2008Article
- MCAK associates with EB1.Oncogene · 2008Article
- Tripin/hSgo2 recruits MCAK to the inner centromere to correct defective kinetochore attachments.The Journal of cell biology · 2007Article
- The interplay of the N- and C-terminal domains of MCAK control microtubule depolymerization activity and spindle assembly.Molecular biology of the cell · 2007Article
- Misorientation and reduced stretching of aligned sister kinetochores promote chromosome missegregation in EB1- or APC-depleted cells.The EMBO journal · 2006Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Traditionally, kinesins have been identified as proteins that use the energy of ATP to translocate along microtubules. However, in the last decade some kinesin-like proteins were found to destabilize microtubule ends. The kinesins that destabilize microtubules are known as "catastrophe kinesins". Analyses of a Xenopus member of the catastrophe kinesins called MCAK/XKCM1 have shown that, in fact, catastrophe kinesins are essential for controlling the distribution of microtubules by inducing their depolymerization. Therefore, unraveling the mechanisms of how microtubule destabilization promoted by these catastrophe kinesins is controlled is essential for understanding how microtubules in a cell are distributed. Here we give an overview of the studies that have focused on the global and local control of microtubule destabilization promoted by MCAK/XKCM1.
Indexed as
Identifiers
16450057What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.