Evidence map›Paper›PMID 16107109›Full record

ReviewTexas Heart Institute journal2005

Amyloid heart disease. New frontiers and insights in pathophysiology, diagnosis, and management.

Walid Hassan, Hani Al-Sergani, Walid Mourad, Rashed Tabbaa

Open access · greenAbstract readReview
In one paragraph

Review in Texas Heart Institute journal, 2005. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 46 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
46citing papers in PubMed, 1 pooled it
2.1field-weighted citation impact, top 14% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

46 citing papers in PubMed, 1 synthesis or guideline pooled it, 125 citations in OpenAlex.

  1. Pooled it
  2. Article
  3. Multimodality Imaging in Cardiac Amyloidosis.Journal of clinical medicine · 2025
    Review
  4. Article
  5. Article
  6. Article
  7. Part I: Cutaneous manifestations of cardiovascular disease.Journal of the American Academy of Dermatology · 2023
    Review
  8. Review
  9. Cardiac Amyloidosis: A Rare TTR Mutation Found in an Asian Female.Journal of cardiovascular development and disease · 2023
    Article
  10. Signal peptide stabilizes folding and inhibits misfolding of serum amyloid A.Protein science : a publication of the Protein Society · 2022
    Article
  11. Article
  12. Review
  13. Review
  14. Review
  15. Article
  16. Review
  17. Article
  18. AL type cardiac amyloidosis: a devastating fatal disease.Journal of community hospital internal medicine perspectives · 2021
    Article
  19. Article
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors at 1 institution in 1 country.

Walid HassanDepartment of Cardiovascular Disease, King Faisal Specialist Hospital and Research Center, Riyadh 11211, Saudi Arabia. hassanw@kfshrc.edu.sa
Hani Al-Sergani
Walid Mourad
Rashed Tabbaa
King Faisal Specialist Hospital & Research Centre · SA

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Amyloidosis comprises a unique group of diseases that share in common the extracellular deposition of insoluble fibrillar proteins in organs and tissues. Cardiovascular amyloidosis can be primary, a part of systemic amyloidosis, or a result of chronic systemic diseases elsewhere in the body. The most common presentations are congestive heart failure-mainly a restrictive infiltrative pattern--and conduction system disturbances. Recent developments in imaging techniques and extracardiac tissue sampling have minimized the need for invasive endomyocardial biopsy for amyloidosis. Despite advances in treatment, the prognosis for patients with amyloidosis is still poor and depends on the underlying disease type. Herein, we present new insights and recent advances in cardiovascular amyloidosis.

Indexed as

AmyloidosisCardiomyopathiesAmyloidAmyloidosis, FamilialCardiomyopathy, RestrictiveEchocardiographyElectrocardiographyHumansMyocardiumPrognosisAmyloid

Identifiers

PMID16107109
PMCPMC1163465
OpenAlexW1945652469

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.