Evidence map›Paper›PMID 16037790›Full record

ArticleOral surgery, oral medicine, oral pathology, oral radiology, and endodontics2005

Pleiotropic mechanisms of virus survival and persistence.

Craig S Miller

Open access · bronzeAbstract readComment
In one paragraph

Article in Oral surgery, oral medicine, oral pathology, oral radiology, and endodontics, 2005. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed
1.0field-weighted citation impact, top 25% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 11 citations in OpenAlex.

  1. Review
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  4. Review
  5. Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

Craig S MillerSection of Oral Medicine, Center for Oral Health Research, College of Dentistry, and Department of Microbiology, Immunology & Molecular Genetics, College of Medicine, University of Kentucky, Lexington 40536-0297, USA.
University of Kentucky · US

Funding

Opportunistic Oral HSV-- Mechanisms of ReactivationR01DE014142 · NIDCR · UNIVERSITY OF KENTUCKY · PI MILLER, CRAIG S · 2001 to 2006
$1.4M
NIDCR NIH HHS R01 DE014142
6 · The paper itself

Abstract

Viruses are enormously efficient infectious agents that have been implicated in causing human disease for centuries. Transmission of these pathogens continues to be from one life form to another in the form of isolated cases, epidemics, and pandemics. Each infection requires entry into a susceptible host, replication, and evasion of the immune system. Viruses are successful pathogens because they target specific cells for their attack, exploit the cellular machinery, and are efficient in circumventing and/or inhibiting key cellular events required of survival. This article reviews some of the advances that have taken place in human virology in the past 50 years, emphasizing mechanisms that contribute to, and are involved with, virus survival and persistence.

Indexed as

ApoptosisCaspase InhibitorsHumansImmunity, CellularInterferonsReceptors, VirusViral ProteinsVirus IntegrationVirus LatencyVirus ReplicationCaspase InhibitorsInterferonsReceptors, VirusViral Proteins

Identifiers

PMID16037790
PMCPMC7118778
OpenAlexW2036775150

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.