Evidence map›Paper›PMID 15939554›Full record

ArticleGene2005

Genomic structure and functional characterization of the promoter region of human IkappaB kinase-related kinase IKKi/IKKvarepsilon gene.

Naizhen Wang, Salahuddin Ahmed, Tariq M Haqqi

Open access · greenAbstract readComparative Study
In one paragraph

Article in Gene, 2005. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.

0numbers the graph read from it
0cells of the map it votes in
11citing papers in PubMed
1.1field-weighted citation impact, top 21% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

11 citing papers in PubMed, 29 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
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  6. Review
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  10. Article
  11. The interferon inducing pathways and the hepatitis C virus.World journal of gastroenterology · 2007
    Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

Naizhen WangDivision of Rheumatic Diseases, Department of Medicine, Case Western Reserve University, 10900 Euclid Avenue, Cleveland, OH 44106, USA.
Salahuddin Ahmed
Tariq M Haqqi
Case Western Reserve University · US

Funding

TRAINING PROGRAM IN MUSCULOSKELETAL RESEARCHT32AR007505 · NIAMS · CASE WESTERN RESERVE UNIVERSITY · PI TRIOLO, RONALD J · 1986 to 2023
$6.3M
PREVENTION OF CARTILAGE DEGRADATION BY GREEN TEAR01AR048782 · NIAMS · CASE WESTERN RESERVE UNIVERSITY · PI HAQQI, TARIQ M · 2002 to 2006
$1.4M
MECHANISM OF INDUCED PROTECTIVE IMMUNITY IN MURINE CIAR01AR044902 · NIAMS · CASE WESTERN RESERVE UNIVERSITY · PI HAQQI, TARIQ M · 1999 to 2002
$635k
Induction Of Apoptosis in Osteosarcoma By Green TeaR21AT002258 · NCCIH · CASE WESTERN RESERVE UNIVERSITY · PI HAQQI, TARIQ M · 2004 to 2005
$446k
NCCIH NIH HHS AT-002258NCCIH NIH HHS R21 AT002258NIAMS NIH HHS AR-07505NIAMS NIH HHS AR-44902NIAMS NIH HHS AR-48782NIAMS NIH HHS R01 AR048782NIAMS NIH HHS T32 AR007505
6 · The paper itself

Abstract

The inducible IkappaB kinase (IKKi/IKKepsilon) is a recently described serine-threonine kinase that activates the transcription factors NFkappaB, interferon regulatory factor-3 (IRF3) and CCAAA/enhancer-binding protein (C/EBPdelta). Several inflammatory agents have been shown to induce the expression of the IKKi gene in macrophages and other cell types but the mechanism is unknown. We have found that the IKKi expression was constitutive in human chondrocytes from OA cartilage and a human chondrocytic cell line C28/I2 but was up-regulated by the inflammatory cytokines TNFalpha or IL-1betain an NFkappaB-dependent manner. To understand the constitutive and inducible expression of the IKKi gene we localized the transcription start site (TSS), cloned and sequenced a 2 kb genomic DNA fragment 5' of the TSS and characterized the putative promoter region (PPR), and identified the motifs therein that are required for basal and cytokine-induced IKKi gene promoter activity. We found that IKKi core promoter was TATA-less and by using PCR generated deletion mutants of the PPR we found that the cis-elements responsible for basal transcriptional activity were located between -51 and -100 bp upstream of the TSS while the cytokine response elements were located distally between -501 and -1000 bp upstream of the TSS. The DNA region containing the cytokine response elements had two kappaB sites as the most relevant regulatory motifs. The results of site-directed mutagenesis revealed that the kappaB site located between -833 and -847 bp upstream of the TSS was biologically functional and required for cytokine-induced IKKi promoter activity in human chondrocytes and HeLa cells. The silence of the other kappaB site (-816/-802) was positional, rather than sequence-specific. Over-expression of NFkappaB p65 mimics the TNFalpha-induced activation of the IKKi promoter. Also the gel shift assay suggested that NFkappaB p65 is responsible for activation of the IKKi promoter. These data for the first time characterize the promoter region and provide further insights into the transcriptional regulation of IKKi in human chondrocytes and other cell types.

Indexed as

Alkaline PhosphataseBase SequenceBinding SitesCell LineChondrocytesElectrophoretic Mobility Shift AssayGene Expression RegulationGPI-Linked ProteinsHeLa CellsHumansI-kappa B KinaseInterleukin-1IsoenzymesLuciferasesMolecular Sequence DataMutationAlkaline Phosphatasealkaline phosphatase, placentalCHUK protein, humanGPI-Linked ProteinsI-kappa B KinaseIKBKB protein, humanIKBKE protein, humanInterleukin-1IsoenzymesLuciferasesNF-kappa BNuclear ProteinsOligonucleotidesProtein Serine-Threonine KinasesRecombinant Fusion ProteinsRNA, MessengerTumor Necrosis Factor-alpha

Identifiers

PMID15939554
PMCPMC1266289
OpenAlexW2037823663

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.