Evidence map›Paper›PMID 15923640›Full record

ArticleMolecular and cellular biology2005

Conditional transgenic system for mouse aurora a kinase: degradation by the ubiquitin proteasome pathway controls the level of the transgenic protein.

Tomokazu Fukuda, Yuji Mishina, Michael P Walker, Richard P DiAugustine

Erratum issuedOpen access · greenAbstract read
In one paragraph

Article in Molecular and cellular biology, 2005. The graph could read no effect estimate from its abstract, so it casts no vote on the map. An erratum has been issued. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
1.0field-weighted citation impact, top 27% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 46 citations in OpenAlex.

  1. Article
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  10. Review
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  12. Augmentation of Smad-dependent BMP signaling in neural crest cells causes craniosynostosis in mice.Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research · 2013
    Article
  13. Aurora A kinase (AURKA) in normal and pathological cell division.Cellular and molecular life sciences : CMLS · 2013
    Review
  14. Article
  15. Article
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

4 authors at 2 institutions in 2 countries.

Tomokazu FukudaHormones and Cancer Group, Laboratory of Molecular Carcinogenesis, National Institute of Environmental Health Sciences, Mail Drop D4-04, Research Triangle Park, NC 27709, USA.
Yuji Mishina
Michael P Walker
Richard P DiAugustine
Laboratory of Molecular Genetics · PLNational Institute of Environmental Health Sciences · US

Funding

Genetics Of Morphogenetic Mechanism During Mouse DevelopZ01ES071003 · NIEHS · NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES · PI MISHINA, YUJI · 1998 to 2008
$4.3M
BIOCHEMICAL MECHANISMS RELATED TO RISK FACTORS OF MAMMARY CARCINOGENESISZ01ES048005 · NIEHS · ENVIRONMENTAL HEALTH SCIENCES · PI DIAUGUSTINE, RICHARD P · 1988 to 2007
$336k
6 · The paper itself

Abstract

Aurora A is a mitotic kinase that localizes to centrosomes. Expression of this protein is normally limited to the mitotic stage (G(2)-M) of the cell cycle, whereas human cancer cells frequently exhibit overexpression of Aurora A protein regardless of the cell cycle stage. In the present study, Aurora A transgenic mouse lines were generated with a new conditional expression system (cytomegalovirus immediate early enhancer-chicken beta-actin hybrid promoter-Z-enhanced green fluorescent protein) in order to analyze the function of this protein. Although transcripts for Aurora A were elevated in multiple organs of the transgenic mice, the corresponding protein was not detected in extracts analyzed by immunoblotting. The treatment of transgenic-derived embryonic fibroblasts (MEF) with proteasome inhibitors markedly increased the protein level of transgenic Aurora A, indicating that the transgenic Aurora A protein is readily degraded in normal mouse tissues. Under the exponential growth conditions of MEF cells, transgenic Aurora A was detected within the mitotic stage of the cell cycle and localized to centrosomes. In contrast, the marker of the transgenic promoter (enhanced green fluorescent protein) was continuously expressed throughout the cell cycle, indicating the constitutive transcription of transgenic mRNA. These results indicate that transgenic Aurora A is protected from degradation within G(2)-M but is immediately degraded after translation in the G(1)-S stage of the cell cycle. The findings obtained with this transgenic model and derived cells support that the transition from protection to degradation by the ubiquitin proteasome system at the end of mitosis is an important step in controlling the level of Aurora A protein during the cell cycle.

Indexed as

Mice, TransgenicAnimalsAurora Kinase AAurora KinasesBrainCell CycleCells, CulturedChickensEmbryo, MammalianEmbryo, NonmammalianFibroblastsHumansKidneyMiceMolecular Sequence DataProteasome Endopeptidase ComplexAurka protein, mouseAurora Kinase AAurora KinasesProteasome Endopeptidase ComplexProteasome InhibitorsProtein Serine-Threonine KinasesRecombinant Fusion ProteinsRNA, MessengerUbiquitin

Identifiers

PMID15923640
PMCPMC1140609
OpenAlexW2097668523

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.