ArticleGenetics1992
The proximal end of mouse chromosome 17: new molecular markers identify a deletion associated with quakingviable.
Article in Genetics, 1992. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
19 citing papers in PubMed, 43 citations in OpenAlex.
- RNA-binding protein quaking: a multifunctional regulator in tumour progression.Annals of medicine · 2025Review
- The Quaking RNA-binding proteins as regulators of cell differentiation.Wiley interdisciplinary reviews. RNA · 2022Review
- Oligodendroglial defects during quakingviable cerebellar development.Developmental neurobiology · 2016Article
- Article
- Solution structure of the QUA1 dimerization domain of pXqua, the Xenopus ortholog of Quaking.PloS one · 2013Article
- Mammalian developmental genetics in the twentieth century.Genetics · 2012Article
- Structural analysis of the quaking homodimerization interface.Journal of molecular biology · 2012Article
- Quaking regulates Hnrnpa1 expression through its 3' UTR in oligodendrocyte precursor cells.PLoS genetics · 2011Article
- Insights into the structural basis of RNA recognition by STAR domain proteins.Advances in experimental medicine and biology · 2010Review
- Rescuing qkV dysmyelination by a single isoform of the selective RNA-binding protein QKI.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2006Article
- A new ENU-induced allele of mouse quaking causes severe CNS dysmyelination.Mammalian genome : official journal of the International Mammalian Genome Society · 2005Article
- Deletion of the Parkin coregulated gene causes male sterility in the quaking(viable) mouse mutant.Proceedings of the National Academy of Sciences of the United States of America · 2004Article
- The neurological mutant quaking(viable) is Parkin deficient.Mammalian genome : official journal of the International Mammalian Genome Society · 2004Article
- A new spontaneous deletion on chromosome 17 including brachyury.Mammalian genome : official journal of the International Mammalian Genome Society · 1997Article
- Neural cell type-specific expression of QKI proteins is altered in quakingviable mutant mice.The Journal of neuroscience : the official journal of the Society for Neuroscience · 1996Article
- Endocytic depletion of L-MAG from CNS myelin in quaking mice.The Journal of cell biology · 1995Article
- Detailed physical and genetic mapping in the region of plasminogen, D17Rp17e, and quaking.Mammalian genome : official journal of the International Mammalian Genome Society · 1993Article
- New molecular markers for the distal end of the t-complex and their relationships to mutations affecting mouse development.Genetics · 1992Article
- Mouse chromosome 17.Mammalian genome : official journal of the International Mammalian Genome Society · 1992Review
Corrections and comments
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Authors and funding
3 authors at 1 institution in 1 country.
Funding
Abstract
Five randomly identified cosmids have been mapped proximal to the Leh66D locus on mouse chromosome 17. Two of these cosmids, Au10 and Au119, map near the neurological mutation quaking. Au119 is deleted in qkviable/qkviable DNA, whereas Au10 is not. Au76 maps to a gene-rich region near the Time locus. The Au76 locus encodes a member of a low copy gene family expressed in embryos, the adult central nervous system and testis. A second member of this family has been mapped to chromosome 15 near c-sis (PDGF-B). At the centromeric end of chromosome 17, Au116 maps near the Tu1 locus, and along with Au217rs identifies a region of unusually high recombinational activity between t-haplotypes and wild-type chromosomes. Au217I and II map to the large inverted repeats found at the proximal end of the wild-type chromosome. In addition, the Au217I and/or II loci encode testis transcripts not expressed from t-haplotypes.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.