Evidence map›Paper›PMID 15599567›Full record

ReviewZeitschrift fur Kardiologie2004

Coronary artery anomalies. Part I: Recent insights from molecular embryology.

Y von Kodolitsch, W D Ito, O Franzen, G K Lund, D H Koschyk, T Meinertz

Abstract readReview
PubMed Publisher
In one paragraph

Review in Zeitschrift fur Kardiologie, 2004. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 9 papers.

0numbers the graph read from it
0cells of the map it votes in
9citing papers in PubMed
1.6field-weighted citation impact, top 16% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

9 citing papers in PubMed, 30 citations in OpenAlex.

  1. Article
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Providing insights into atherosclerosis: A case of multivessel coronary artery disease with an anomalous straightforward vessel spared from atherosclerosis.The International journal of angiology : official publication of the International College of Angiology, Inc · 2010
    Article
  8. Coronary vessel development and insight towards neovascular therapy.International journal of experimental pathology · 2009
    Review
  9. Single coronary artery with anomalous origin from the right sinus Valsalva.Clinical research in cardiology : official journal of the German Cardiac Society · 2006
    Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 2 institutions in 1 country.

Y von KodolitschClinic of Internal Medicine III, University Hospital Hamburg-Eppendorf, Martinistrasse 52, 20246 Hamburg, Germany. kodolitsch@uke.uni-hamburg.de
W D Ito
O Franzen
G K Lund
D H Koschyk
T Meinertz
Universität Hamburg · DEUniversity Medical Center Hamburg-Eppendorf · DE

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Congenital anomalies of the coronary arteries occur in 0.2-1.2% of the general population and may cause substantial cardiovascular morbidity and mortality. We review some of the advances that have been made both, in the understanding of the embryonic development of the coronary arteries (part I) and in the clinical diagnosis and management of their anomalies (part II). In this first part of our review we elucidate basic mechanisms of coronary vasculogenesis, angiogenesis and embryonic arteriogenesis. Moreover, we review the role of cellular progenitors such as epicardium-derived cells, cardiac neural crest cells and cells of the peripheral conduction system. Then we discuss the role of growths factors (such as FGV, HIF 1, PDGF B, TGFbeta1, VEGF, and VEGFR-2) and genes (such as FOG-2, VCAM-1, Bves, and RALDH2) at different states of coronary development. and we discuss the role of the cardiac neural crest in the concurrence of coronary anomalies with aortic root malformations. This part of the article is designed to review major determinants of coronary vascular development to provide a better understanding of the multiplicity of options and mechanisms that may give rise to coronary anomaly. To this end, we highlight results from experiments that provide insight in mechanisms of coronary malformation.

Indexed as

AnimalsCoronary Vessel AnomaliesCoronary VesselsEndothelium, VascularFemaleGene Expression RegulationGestational AgeGrowth SubstancesHumansInfant, NewbornNeovascularization, PhysiologicPregnancyQuailStem CellsGrowth Substances

Identifiers

PMID15599567
OpenAlexW4514158

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.