Evidence map›Paper›PMID 15542838›Full record

ArticleMolecular and cellular biology2004

Bcr-Abl-mediated protection from apoptosis downstream of mitochondrial cytochrome c release.

Paula B Deming, Zachary T Schafer, Jessica S Tashker, Malia B Potts, Mohanish Deshmukh, Sally Kornbluth

Open access · greenAbstract read
In one paragraph

Article in Molecular and cellular biology, 2004. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 25 papers.

0numbers the graph read from it
0cells of the map it votes in
25citing papers in PubMed
1.5field-weighted citation impact, top 18% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

25 citing papers in PubMed, 60 citations in OpenAlex.

  1. Styrylquinazoline derivatives as ABL inhibitors selective for different DFG orientations.Journal of enzyme inhibition and medicinal chemistry · 2023
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  12. Cardiotoxicity associated with targeted cancer therapies.Molecular and clinical oncology · 2016
    Article
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  15. Review
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  17. Review
  18. Article
  19. Mitochondrial control of caspase-dependent and -independent cell death.Cellular and molecular life sciences : CMLS · 2010
    Review
  20. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

6 authors at 3 institutions in 1 country.

Paula B DemingDepartment of Pharmacology and Cancer Biology, Duke University Medical Center, Box 3813, Durham, NC 27710, USA.
Zachary T Schafer
Jessica S Tashker
Malia B Potts
Mohanish Deshmukh
Sally Kornbluth
Duke Medical Center · USDuke University Hospital · USUniversity of North Carolina at Chapel Hill · US

Funding

Mechanisms of Increasing Apoptosis Regulation in Developing and Mature NeuronsR01NS042197 · NINDS · UNIV OF NORTH CAROLINA CHAPEL HILL · PI DESHMUKH, MOHANISH P · 2001 to 2011
$3.5M
NCI NIH HHS R01 CA102702NINDS NIH HHS R01 NS042197NINDS NIH HHS R01 NS42197
6 · The paper itself

Abstract

Bcr-Abl, activated in chronic myelogenous leukemias, is a potent cell death inhibitor. Previous reports have shown that Bcr-Abl prevents apoptosis through inhibition of mitochondrial cytochrome c release. We report here that Bcr-Abl also inhibits caspase activation after the release of cytochrome c. Bcr-Abl inhibited caspase activation by cytochrome c added to cell-free lysates and prevented apoptosis when cytochrome c was microinjected into intact cells. Bcr-Abl acted posttranslationally to prevent the cytochrome c-induced binding of Apaf-1 to procaspase 9. Although Bcr-Abl prevented interaction of endogenous Apaf-1 with the recombinant prodomain of caspase 9, it did not affect the association of endogenous caspase 9 with the isolated Apaf-1 caspase recruitment domain (CARD) or Apaf-1 lacking WD-40 repeats. These data suggest that Apaf-1 recruitment of caspase 9 is faulty in the presence of Bcr-Abl and that cytochrome c/dATP-induced exposure of the Apaf-1 CARD is likely defective. These data provide a novel locus of Bcr-Abl antiapoptotic action and suggest a distinct mechanism of apoptosomal inhibition.

Indexed as

ApoptosisAnimalsApoptotic Protease-Activating Factor 1Blotting, WesternCaspase 9CaspasesCell LineCell Line, TumorCells, CulturedCytochromes cCytosolEnzyme ActivationFibroblastsFusion Proteins, bcr-ablGlutathioneGreen Fluorescent ProteinsAPAF1 protein, humanApaf1 protein, mouseApaf1 protein, ratApoptotic Protease-Activating Factor 1CASP9 protein, humanCasp9 protein, mouseCasp9 protein, ratCaspase 9CaspasesCytochromes cFusion Proteins, bcr-ablGlutathioneGreen Fluorescent ProteinsPhosphotyrosineProteinsRecombinant Proteins

Identifiers

PMID15542838
PMCPMC529043
OpenAlexW2105209136

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.