ReviewCurrent hypertension reports2004
Evolution of diagnostic criteria for primary aldosteronism: why is it more common in "drug-resistant" hypertension today?
Review in Current hypertension reports, 2004. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
10 citing papers in PubMed.
- ACE Inhibition to Distinguish Low-Renin Hypertension From Primary Aldosteronism.Hypertension (Dallas, Tex. : 1979) · 2025Article
- The Spectrum of Dysregulated Aldosterone Production: An International Human Physiology Study.The Journal of clinical endocrinology and metabolism · 2024Article
- The Unrecognized Prevalence of Primary Aldosteronism: A Cross-sectional Study.Annals of internal medicine · 2020Article
- Urotensin II Exerts Pressor Effects By Stimulating Renin And Aldosterone Synthase Gene Expression.Scientific reports · 2017Article
- Individualizing antihypertensive combination therapies: clinical and hemodynamic considerations.Current hypertension reports · 2014Review
- Resistant hypertension and undiagnosed primary hyperaldosteronism detected by use of a computerized database.Journal of clinical hypertension (Greenwich, Conn.) · 2011Article
- Clinical approach in treatment of resistant hypertension.Integrated blood pressure control · 2009Article
- Familial mineralocorticoid induced hypertension in the sultanate of oman.Sultan Qaboos University medical journal · 2008Article
- Primary aldosteronism: diagnosis and treatment.Journal of clinical hypertension (Greenwich, Conn.) · 2006Review
- Treatment of resistant hypertension.Current hypertension reports · 2005Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
1 author.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The recent "epidemic" of primary aldosteronism reported in the literature is most likely related to the widespread acceptance that with easy access to accurate measurements of renin and aldosterone, it is no longer necessary to wait until hypokalemia has become profound before embarking on diagnostic testing to attempt to ferret out this most common cause of "essential" hypertension. This is especially true for those who are now classified as "drug resistant" using today's popular drugs, which are particularly ineffective in lowering blood pressure in primary aldosteronism and its variants. Understanding the physiologic consequences of a slowly increasing aldosterone production by autonomous cells will help both the family practitioner and the specialist understand the role of the aldosterone renin ratio (ARR) in the care of the hypertensive patient. In addition, the increasing number of specific genetic mutations that drive sodium retention and lead to low levels of renin activity and familial hypertension must be incorporated into the routine evaluation and care of hypertensive patients and their families.
Indexed as
Identifiers
15527695What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.