ArticleThe Biochemical journal2005
Characterization of the molecular forms of glutathione S-transferase P1 in human gastric cancer cells (Kato III) and in normal human erythrocytes.
Article in The Biochemical journal, 2005. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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Who cites it
5 citing papers in PubMed, 17 citations in OpenAlex.
- Oxidative Imbalance and Kidney Damage in Cafeteria Diet-Induced Rat Model of Metabolic Syndrome: Effect of Bergamot Polyphenolic Fraction.Antioxidants (Basel, Switzerland) · 2019Article
- Characterization of affinity-purified isoforms of Acinetobacter calcoaceticus Y1 glutathione transferases.TheScientificWorldJournal · 2014Article
- The effects of intralaboratory modifications to media composition and cell source on the expression of pharmaceutically relevant transporters and metabolizing genes in the Caco-2 cell line.Journal of pharmaceutical sciences · 2012Article
- GST pi modulates JNK activity through a direct interaction with JNK substrate, ATF2.Protein science : a publication of the Protein Society · 2011Article
- Tyrosine phosphorylation of the human glutathione S-transferase P1 by epidermal growth factor receptor.The Journal of biological chemistry · 2009Article
Corrections and comments
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Authors and funding
3 authors at 1 institution in 1 country.
Funding
Abstract
GSTP1 (glutathione S-transferase pi) is involved in stress responses and in cellular proliferation pathways as an inhibitor of JNK (c-Jun N-terminal kinase). It has been proposed that monomeric GSTP1 functions as a JNK inhibitor. All of the studies to date have been performed using rodent cells, and it is unclear if monomeric GSTP1 exists in human cells. Monomeric GSTP1 was sought in human gastric cancer cells (Kato III) and in normal human erythrocytes using gel filtration, ELISA and Western blots. Monomeric GSTP1 was found in conditioned medium, in cytosol of Kato III cells and in cytosol of erythrocytes. GSTP1 subunits from Kato III cells and erythrocytes were heterogeneous when analysed by MALDI-TOF (matrix-assisted laser-desorption ionization-time-of-flight) MS, suggesting that there were post-translational modifications to GSTP1. One post-translational modification, phosphorylation of a serine residue in the C-terminal portion of GSTP1 where JNK binds, was identified in GSTP1 purified from Kato III cells, but not in GSTP1 purified from human erythrocytes. Therefore normal and malignant human cells contain GSTP1 monomers with post-translational modifications, and it is likely that GSTP1 monomers regulate JNK activity in human cells in the same manner as in rodent cells.
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Registered trials
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