ArticleVirchows Archiv : an international journal of pathology2004
Monosomy 9q and trisomy 16q in a case of congenital solitary infantile myofibromatosis.
Article in Virchows Archiv : an international journal of pathology, 2004. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 6 papers.
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Who cites it
6 citing papers in PubMed, 28 citations in OpenAlex.
- [Left mandibular infantile myofibromatosis: a case report].Hua xi kou qiang yi xue za zhi = Huaxi kouqiang yixue zazhi = West China journal of stomatology · 2020Article
- Myofibroma as a Rapidly Growing Gingival Mass in a 4-year-old Boy: a Case Report.Journal of dentistry (Shiraz, Iran) · 2018Article
- Solitary infantile myofibromatosis in the bones of the upper extremities: Two rare cases and a review of the literature.Oncology letters · 2013Article
- Infantile myofibroma eroding into the frontal bone: a case report and review of its histopathologic differential diagnosis.Case reports in pediatrics · 2012Article
- Solitary infantile myofibromatosis of the cranial vault: case report.Child's nervous system : ChNS : official journal of the International Society for Pediatric Neurosurgery · 2011Article
- Infantile myofibromatosis of the iliac bone.Clinical cases in mineral and bone metabolism : the official journal of the Italian Society of Osteoporosis, Mineral Metabolism, and Skeletal DiseasesArticle
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Authors and funding
6 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Although infantile myofibromatosis (IM) is the most common fibrous proliferation of infancy, many aspects of this benign lesion have not been explored. IM histogenesis is still poorly understood, despite immunohistochemical staining and ultrastructural features that suggest a myofibroblastic origin. IM diagnosis is often made difficult by the predominance of small primitive spindle cells over myofibroblasts and the presence of intravascular growth. Genetic information is scarce, with only one karyotyped case. Here we describe a case of solitary IM discovered at birth in an otherwise healthy girl. The tumor was well circumscribed, arranged in nodules and made up of ovoid cells without atypia, in a myxoid background. Immunohistochemical evaluation indicated a myofibroblastic differentiation. The cytogenetic and fluorescence in situ hybridization analyses revealed an abnormal chromosome 9, derived from an unbalanced whole-arm translocation between chromosomes 9 and 16. On both chromosomes, the breakpoints were located in the pericentric heterochromatic region. This clonal abnormality has not been reported in other tumors and is different from the chromosome 6q deletion reported in the single previous reported IM karyotype.
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