ArticleMycopathologia2004
Molecular typing study of the Microsporum canis strains isolated from an outbreak of tinea capitis in a school.
Article in Mycopathologia, 2004. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 11 papers.
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Who cites it
11 citing papers in PubMed, 40 citations in OpenAlex.
- Antifungal activity ofFrontiers in microbiology · 2024Article
- Emerging Animal-Associated Fungal Diseases.Journal of fungi (Basel, Switzerland) · 2022Review
- FSH1 regulates the phenotype and pathogenicity of the pathogenic dermatophyte Microsporum canis.International journal of molecular medicine · 2019Article
- Outbreaks And Epidemics Of Superficial Dermatophytosis Due ToClinical, cosmetic and investigational dermatology · 2019Review
- Investigating Clinical Issues by Genotyping of Medically Important Fungi: Why and How?Clinical microbiology reviews · 2017Review
- Pathogenic fungus Microsporum canis activates the NLRP3 inflammasome.Infection and immunity · 2014Article
- Microsporum canis infection in three familial cases with tinea capitis and tinea corporis.Mycopathologia · 2013Article
- Microsatellite-primed PCR and random primer amplification polymorphic DNA for the identification and epidemiology of dermatophytes.European journal of clinical microbiology & infectious diseases : official publication of the European Society of Clinical Microbiology · 2013Article
- Single-step PCR using (GACA)4 primer: utility for rapid identification of dermatophyte species and strains.Journal of clinical microbiology · 2008Article
- High-frequency intragenomic heterogeneity of the ribosomal DNA intergenic spacer region in Trichophyton violaceum.Eukaryotic cell · 2008Article
- Strain differentiation of dermatophytes.MycopathologiaReview
Corrections and comments
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Authors and funding
5 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Tinea capitis is a dermatophyte infection of the scalp that occurs most often in prepubescent children. Tinea capitis may be transmitted by shared use of contaminated hairbrush, by contact with fomites or by direct physical contact with an infected person. Occasionally, outbreak of tinea capitis would happen under some special conditions. Last year, we found an outbreak of tinea capitis in a school due to Microsporum canis. In epidemiological study, we performed the prevalence survey to all of the exposed persons by physical examinations and mycological laboratory tests, including KOH preparation and fungal cultures. We also investigated the environment in the school. In molecular typing study of the M. canis isolated from patients and the environment, random primer amplification polymorphic DNA (RAPD) method, the specific amplification of subrepeat element in the ribosomal DNA nontranscribed spacer (NTS), and the analysis of DNA sequence in the intertranscribed spacer (ITS) of rDNA were performed. The total number of exposed children was seventy-one, among them forty-two were attacked by tinea capitis. The ratio between boy and girl was 13:1. The ages of the patients was ranged from 3.5 years old to 10 years old. Four patients bred cat or dog as pet. Most patients appeared noninflammatory type of tinea capitis and several patients were inflammatory type. Under microscopic examination the invaded hair were all ectothrix. The pathogens isolated from these patients were M. canis. And we also isolated M. canis from the carpet and the pillowcase in the school. The patterns of total strains of M. canis in the RAPD method and PCR amplification of the rDNA NTS region study were identical, and the isolates from patients and the environment contained the same DNA sequences in the ITS region. The outbreak of tinea capitis was caused by M. canis. The M. canis isolated from patients and from the environment were probably the same origin.
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