ReviewJournal of bioenergetics and biomembranes2003
Targeting mechanisms of high voltage-activated Ca2+ channels.
Review in Journal of bioenergetics and biomembranes, 2003. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
17 citing papers in PubMed.
- Functional tuning of Vascular L-type CaFrontiers in physiology · 2022Review
- Spinocerebellar ataxia type 6 protein aggregates cause deficits in motor learning and cerebellar plasticity.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2015Article
- How voltage-gated calcium channels gate forms of homeostatic synaptic plasticity.Frontiers in cellular neuroscience · 2014Review
- Ion channel remodeling in vascular smooth muscle during hypertension: Implications for novel therapeutic approaches.Pharmacological research · 2013Review
- Synaptic NMDA receptor-dependent Ca²⁺ entry drives membrane potential and Ca²⁺ oscillations in spinal ventral horn neurons.PloS one · 2013Article
- Functional coupling of the metabotropic glutamate receptor, InsP3 receptor and L-type Ca2+ channel in mouse CA1 pyramidal cells.The Journal of physiology · 2012Article
- Ion flux dependent and independent functions of ion channels in the vertebrate heart: lessons learned from zebrafish.Stem cells international · 2012Article
- Delayed postnatal loss of P/Q-type calcium channels recapitulates the absence epilepsy, dyskinesia, and ataxia phenotypes of genomic Cacna1a mutations.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2011Article
- The calcium channel beta2 (CACNB2) subunit repertoire in teleosts.BMC molecular biology · 2008Article
- straightjacket is required for the synaptic stabilization of cacophony, a voltage-gated calcium channel alpha1 subunit.The Journal of cell biology · 2008Article
- Facilitation versus depression in cultured hippocampal neurons determined by targeting of Ca2+ channel Cavbeta4 versus Cavbeta2 subunits to synaptic terminals.The Journal of cell biology · 2007Article
- Importance of voltage-dependent inactivation in N-type calcium channel regulation by G-proteins.Pflugers Archiv : European journal of physiology · 2007Article
- Maturation of ribbon synapses in hair cells is driven by thyroid hormone.The Journal of neuroscience : the official journal of the Society for Neuroscience · 2007Article
- Bioenergetic analysis of cerebellar granule neurons undergoing apoptosis by potassium/serum deprivation.Cell death and differentiation · 2006Article
- Ca1.2 and CaV1.3 neuronal L-type calcium channels: differential targeting and signaling to pCREB.The European journal of neuroscience · 2006Article
- Vascular calcium channels and high blood pressure: pathophysiology and therapeutic implications.Vascular pharmacology · 2006Review
- Calcium channels: unanswered questions.Journal of bioenergetics and biomembranes · 2003Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
7 authors.
Funding
Abstract
Functional voltage-dependent Ca2+ channel complexes are assembled by three to four subunits: alpha1, beta, alpha2delta subunits (C. Leveque et al., 1994, J. Biol Chem. 269, 6306-6312; M. W. McEnery et al., 1991, Proc. Natl. Acad. Sci. U.S.A. 88, 11095-11099) and at least in muscle cells also y subunits (B. M. Curtis and W. A. Catterall, 1984, Biochemistry 23, 2113-2118). Ca2+ channels mediate the voltage-dependent Ca2+ influx in subcellular compartments, triggering such diverse processes as neurotransmitter release, dendritic action potentials, excitation-contraction, and excitation-transcription coupling. The targeting of biophysically defined Ca2+ channel complexes to the correct subcellular structures is, thus, critical to proper cell and physiological functioning. Despite their importance, surprisingly little is known about the targeting mechanisms by which Ca2+ channel complexes are transported to their site of function. Here we summarize what we know about the targeting of Ca2+ channel complexes through the cell to the plasma membrane and subcellular structures.
Indexed as
Identifiers
15000523What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.