ArticleBiological cybernetics1992
Control of rabbit nictitating membrane movements. II. Analysis of the relation of motoneuron activity to behavior.
Article in Biological cybernetics, 1992. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
5 citing papers in PubMed, 9 citations in OpenAlex.
- An agonist-antagonist cerebellar nuclear system controlling eyelid kinematics during motor learning.Frontiers in neuroanatomy · 2012Article
- Neural circuitry and plasticity mechanisms underlying delay eyeblink conditioning.Learning & memory (Cold Spring Harbor, N.Y.) · 2011Review
- Recruitment in retractor bulbi muscle during eyeblink conditioning: EMG analysis and common-drive model.Journal of neurophysiology · 2009Article
- Metabotropic glutamate receptor activation in cerebellar Purkinje cells as substrate for adaptive timing of the classically conditioned eye-blink response.The Journal of neuroscience : the official journal of the Society for Neuroscience · 1996Article
- Control of rabbit nictitating membrane movements. I. A computer model of the retractor bulbi muscle and the associated orbital mechanics.Biological cybernetics · 1992Article
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Authors and funding
2 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The objective of this study is to understand more precisely the relationship of motoneuron activity to movements of the rabbit nictitating membrane (NM). We use a model of the oculomotor plant to investigate what NM movements are generated by a given pattern of neural input and what inputs are required to generate particular NM movements. Simulated peak NM extensions can occur well over 50 ms after the end of motoneuron activity. The neural input required for the model to generate full amplitude NM extension responses is more consistent with single accessory abducens unit recordings from awake rabbits than recordings from anesthetized rabbits. An initial high frequency burst of neural activity followed by a rapid decay is required for simulations of conditioned responses (CRs) trained at a 125 ms interstimulus interval (ISI). For CRs trained with a 250 ms ISI, a more slowly rising and decaying neural activity is required. Model simulations show that the linear correlation between the shape of histogrammed motoneuron activity and the shape of NM movements can be high for long duration responses (> 400 ms) but is low for short duration responses (< 200 ms). Simulations are also consistent with the hypothesis that NM retraction is generally passive.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.