Presentation of a self-peptide in two distinct conformations by a disease-associated HLA-B27 subtype.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

5 citing papers in PubMed.

1. Ankylosing spondylitis: etiology, pathogenesis, and treatments.Bone research · 2019
   Article
2. Pathophysiology of spondyloarthritis.Current rheumatology reports · 2011
   Article
3. Predicting important residues and interaction pathways in proteins using Gaussian Network Model: binding and stability of HLA proteins.PLoS computational biology · 2010
   Article
4. Directed evolution of human T cell receptor CDR2 residues by phage display dramatically enhances affinity for cognate peptide-MHC without increasing apparent cross-reactivity.Protein science : a publication of the Protein Society · 2006
   Article
5. Thermodynamics of T-cell receptor-peptide/MHC interactions: progress and opportunities.Journal of molecular recognition : JMR
   Review

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

Authors and funding

1 author.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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