ArticleThe Journal of clinical investigation1992
Selective induction of rheumatoid factors by superantigens and human helper T cells.
Article in The Journal of clinical investigation, 1992. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
10 citing papers in PubMed, 50 citations in OpenAlex.
- Incorporating mucosal-associated invariant T cells into the pathogenesis of chronic liver disease.World journal of gastroenterology · 2021Review
- Identification of a unique TCR repertoire, consistent with a superantigen selection process in Children with Multi-system Inflammatory Syndrome.bioRxiv : the preprint server for biology · 2020Article
- Staphylococcal superantigens and T cell expansions in Wegener's granulomatosis.Clinical and experimental immunology · 2003Article
- Differential requirements for induction of total immunoglobulin and physiological rheumatoid factor production by human peripheral blood B cells.Clinical and experimental immunology · 2001Article
- VH3-21 B cells escape from a state of tolerance in rheumatoid arthritis and secrete rheumatoid factor.Molecular medicine (Cambridge, Mass.) · 1995Article
- Increased levels of serum IgM antibody to staphylococcal enterotoxin B in patients with rheumatoid arthritis.Annals of the rheumatic diseases · 1995Article
- Staphylococcus aureus Cowan I-induced human immunoglobulin responses: preferential IgM rheumatoid factor production and VH3 mRNA expression by protein A-binding B cells.Journal of clinical immunology · 1995Article
- Induction of B cell apoptosis by TH0, but not TH2, CD4+ T cells.The Journal of clinical investigation · 1995Article
- Bacterial and viral superantigens: roles in autoimmunity?Annals of the rheumatic diseases · 1993Review
- Role of protein glycosylation in immune regulation.Annals of the rheumatic diseases · 1993Review
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
3 authors at 1 institution in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Production of autoantibodies specific for the Fc region of autologous IgG, called rheumatoid factors (RF), is a characteristic finding in patients with rheumatoid arthritis (RA). To study the requirements regulating the synthesis of these autoantibodies, we have cloned human helper T cells and co-cultured them with purified B cells. To mimic cognate T-B cell interaction, we have used bacterial superantigens that function by cross-linking HLA molecules on the B cell with selected T cell receptor (TCR) molecules expressing a particular polymorphism of the V beta gene segment. Data presented here demonstrate that the staphylococcal enterotoxin D (SE D), but not other bacterial superantigens, exhibits an ability to induce IgM, IgG, and especially RF production, in B cells from RA patients and normal individuals. Comparison with the polyclonal antibody production in B cell cultures driven by anti-CD3-stimulated T cell clones confirmed that SE D shifted the repertoire of secreted antibodies toward immunoglobulins with Fc binding specificity, suggesting that SE D preferentially stimulates RF+ B lymphocytes. B cells with the potential to secrete RF were highly frequent in RA patients, requiring as few as 150 peripheral B cells/culture to detect RF in the culture supernatants. SE D-induced RF synthesis was strictly dependent on the presence of selected CD4+T helper cells and required a direct membrane contact between B cells and T helper cells. Here, we propose a model that SE D selectively induces RF production depending on the availability of SE D responsive T cells in the TCR repertoire of the responder.
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