From cilia to cyst.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

60 citing papers in PubMed.

1. Broadening horizons: Pathogenesis and therapeutics of renal ciliopathies.Journal of cell communication and signaling · 2026
   Review
2. The CPLANE protein Fuzzy regulates ciliogenesis by suppressing actin polymerization at the base of the primary cilium via p190A RhoGAP.Development (Cambridge, England) · 2024
   Article
3. Primary cilium in kidney development, function and disease.Frontiers in endocrinology · 2022
   Review
4. Polycystin-2 (TRPP2) regulates primary cilium length in LLC-PK1 renal epithelial cells.Frontiers in physiology · 2022
   Article
5. Nephronophthisis-Pathobiology and Molecular Pathogenesis of a Rare Kidney Genetic Disease.Genes · 2021
   Review
6. A genetic screen in Drosophila reveals an unexpected role for the KIP1 ubiquitination-promoting complex in male fertility.PLoS genetics · 2020
   Article
7. Ciliary Genes in Renal Cystic Diseases.Cells · 2020
   Review
8. The pathobiology of polycystic kidney disease from a metabolic viewpoint.Nature reviews. Nephrology · 2019
   Review
9. Sexual Dimorphism and Sex Differences inGenetics · 2018
   Article
10. Zebrafish as a Model for Drug Screening in Genetic Kidney Diseases.Frontiers in pediatrics · 2018
    Review
11. Ciliary Mechanisms of Cyst Formation in Polycystic Kidney Disease.Cold Spring Harbor perspectives in biology · 2017
    Review
12. The Genetic and Cellular Basis of Autosomal Dominant Polycystic Kidney Disease-A Primer for Clinicians.Frontiers in pediatrics · 2017
    Review
13. Many Genes-One Disease? Genetics of Nephronophthisis (NPHP) and NPHP-Associated Disorders.Frontiers in pediatrics · 2017
    Review
14. Hepatorenal fibrocystic diseases in children.Pediatric nephrology (Berlin, Germany) · 2016
    Article
15. Mechanical properties of a primary cilium as measured by resonant oscillation.Biophysical journal · 2015
    Article
16. Stem cells and fluid flow drive cyst formation in an invertebrate excretory organ.eLife · 2015
    Article
17. DCDC2 mutations cause a renal-hepatic ciliopathy by disrupting Wnt signaling.American journal of human genetics · 2015
    Article
18. Autosomal recessive polycystic kidney disease: a hepatorenal fibrocystic disorder with pleiotropic effects.Pediatrics · 2014
    Review
19. Meckel-Gruber syndrome and the role of primary cilia in kidney, skeleton, and central nervous system development.Organogenesis · 2014
    Review
20. Inversin modulates the cortical actin network during mitosis.American journal of physiology. Cell physiology · 2013
    Article

Corrections and comments

• Comment on
  Mutations in a novel gene, NPHP3, cause adolescent nephronophthisis, tapeto-retinal degeneration and hepatic fibrosis.2003
• Comment on
  Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination.2003

Authors and funding

2 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as