ArticleNature genetics2003
Genetic background is an important determinant of metastatic potential.
Article in Nature genetics, 2003. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 46 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
46 citing papers in PubMed.
- Investigating the role of UBASH3B in cancer: structural relevance, physiological functions, and therapeutic possibilities.Journal of experimental & clinical cancer research : CR · 2025Review
- Impact ofCancers · 2024Article
- High leukocyte mitochondrial DNA copy number contributes to poor prognosis in breast cancer patients.BMC cancer · 2023Article
- Role of germline variants in the metastasis of breast carcinomas.Oncotarget · 2022Article
- Genetic relationship analysis and molecular fingerprint identification of the tea germplasms from Guangxi Province, China.Breeding science · 2021Article
- Molecular principles of metastasis: a hallmark of cancer revisited.Signal transduction and targeted therapy · 2020Review
- A Brief Overview of the Paradoxical Role of Glucocorticoids in Breast Cancer.Breast cancer : basic and clinical research · 2020Review
- Cancer stem cells as key drivers of tumour progression.Journal of biomedical science · 2018Review
- Article
- MicroRNA-145-5p inhibits gastric cancer invasiveness through targeting N-cadherin and ZEB2 to suppress epithelial-mesenchymal transition.OncoTargets and therapy · 2016Article
- [Regulation Mechanism of MTA3 in the Apoptosis of NSCLC Cells].Zhongguo fei ai za zhi = Chinese journal of lung cancer · 2015Article
- The role of individual inheritance in tumor progression and metastasis.Journal of molecular medicine (Berlin, Germany) · 2015Review
- Abnormal expression of FLOT1 correlates with tumor progression and poor survival in patients with non-small cell lung cancer.Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine · 2014Article
- Pttg1/securin is required for the branching morphogenesis of the mammary gland and suppresses mammary tumorigenesis.Proceedings of the National Academy of Sciences of the United States of America · 2014Article
- Overexpression of MTA3 Correlates with Tumor Progression in Non-Small Cell Lung Cancer.PloS one · 2013Article
- Seed, soil, and beyond: The basic biology of brain metastasis.Surgical neurology international · 2013Article
- Animal models and conserved processes.Theoretical biology & medical modelling · 2012Article
- A methodological approach to unravel organ-specific breast cancer metastasis.Journal of mammary gland biology and neoplasia · 2012Review
- Behavior of seeds and soil in the mechanism of metastasis: a deeper understanding.Cancer science · 2012Review
- Hypoxia-inducible factor 1α promotes primary tumor growth and tumor-initiating cell activity in breast cancer.Breast cancer research : BCR · 2012Article
Corrections and comments
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Authors and funding
3 authors.
Funding
Abstract
PubMed holds no abstract for this paper.
Indexed as
Identifiers
12721549What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.