ArticleMolecular and cellular biology2003
Zinc finger protein sall2 is not essential for embryonic and kidney development.
Article in Molecular and cellular biology, 2003. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
20 citing papers in PubMed, 43 citations in OpenAlex.
- Identification of Pappa and Sall3 as Gli3 direct target genes acting downstream of cilia signaling in corticogenesis.Cerebral cortex (New York, N.Y. : 1991) · 2024Article
- Sall genes regulate hindlimb initiation in mouse embryos.Genetics · 2024Article
- The Sall2 transcription factor promotes cell migration regulating focal adhesion turnover and integrin β1 expression.Frontiers in cell and developmental biology · 2022Article
- SALL Proteins; Common and Antagonistic Roles in Cancer.Cancers · 2021Review
- Streamlined computational pipeline for genetic background characterization of genetically engineered mice based on next generation sequencing data.BMC genomics · 2019Article
- SALL2 represses cyclins D1 and E1 expression and restrains G1/S cell cycle transition and cancer-related phenotypes.Molecular oncology · 2018Article
- Sall2 is required for proapoptotic Noxa expression and genotoxic stress-induced apoptosis by doxorubicin.Cell death & disease · 2015Article
- Mutation of SALL2 causes recessive ocular coloboma in humans and mice.Human molecular genetics · 2014Article
- Molecular regulation of kidney development.Anatomy & cell biology · 2013Article
- Sall4 Is Transiently Expressed in the Caudal Wolffian Duct and the Ureteric Bud, but Dispensable for Kidney Development.PloS one · 2013Article
- Mammalian kidney development: principles, progress, and projections.Cold Spring Harbor perspectives in biology · 2012Review
- The role of HSAL (SALL) genes in proliferation and differentiation in normal hematopoiesis and leukemogenesis.Transfusion · 2011Article
- The role of stem cell factor SALL4 in leukemogenesis.Critical reviews in oncogenesis · 2011Review
- Sall2 is a novel p75NTR-interacting protein that links NGF signalling to cell cycle progression and neurite outgrowth.The EMBO journal · 2009Article
- Sall genes regulate region-specific morphogenesis in the mouse limb by modulating Hox activities.Development (Cambridge, England) · 2009Article
- Sall1, sall2, and sall4 are required for neural tube closure in mice.The American journal of pathology · 2008Article
- A transcriptional program mediating entry into cellular quiescence.PLoS genetics · 2007Article
- Tissue microarray profiling of primary and xenotransplanted synovial sarcomas demonstrates the immunophenotypic similarities existing between SYT-SSX fusion gene confirmed, biphasic, and monophasic fibrous variants.Virchows Archiv : an international journal of pathology · 2006Article
- Loss of the Sall3 gene leads to palate deficiency, abnormalities in cranial nerves, and perinatal lethality.Molecular and cellular biology · 2004Article
- p150(Sal2) is a p53-independent regulator of p21(WAF1/CIP).Molecular and cellular biology · 2004Article
Corrections and comments
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Authors and funding
8 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
SALL/Sall is a mammalian homolog of the Drosophila region-specific homeotic gene spalt (sal), and heterozygous mutations in SALL1 in humans lead to Townes-Brocks syndrome. We earlier reported that mice deficient in Sall1 die in the perinatal period and that kidney agenesis or severe dysgenesis are present. We have now generated mice lacking Sall2, another Sall family gene. Although Sall2 is expressed mostly in an overlapping fashion versus that of Sall1, Sall2-deficient mice show no apparent abnormal phenotypes. Morphology and gene expression patterns of the mutant kidney were not affected. Mice lacking both Sall1 and Sall2 show kidney phenotypes comparable to those of Sall1 knockout, thereby demonstrating the dispensable roles of Sall2 in embryonic and kidney development.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.