Evidence map›Paper›PMID 12223103›Full record

ReviewArthritis research2002

Cartilage-specific autoimmunity in animal models and clinical aspects in patients - focus on relapsing polychondritis.

Ann-Sofie Hansson, Rikard Holmdahl

Abstract readReview
In one paragraph

Review in Arthritis research, 2002. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 4 papers.

0numbers the graph read from it
0cells of the map it votes in
4citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

4 citing papers in PubMed.

  1. Review
  2. CD10Frontiers in immunology · 2020
    Article
  3. In search of chondrocyte-specific antigen.Central-European journal of immunology · 2018
    Article
  4. Article
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors.

Ann-Sofie HanssonDepartment of Clinical Immunology, Sahlgrenska University Hospital, Gothenburg, Sweden. ann-sofie.hansson@vgregion.se
Rikard Holmdahl

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Relapsing polychondritis is an autoimmune disease in which an inappropriate immune response destroys cartilage. Cartilage of the ears, larynx and nose rather than spine and joint cartilage is affected by a chronic relapsing and erosive inflammation. Several animal models for relapsing polychondritis have been published in which immunization with various cartilage proteins induces a variety of chondritis symptoms that mimic those seen in patients. In this review we describe the collagens, matrilin-1 and cartilage oligomeric matrix protein as potential autoantigens able to trigger the tissue-specific immune response seen both in patients and in animal models for relapsing polychondritis and related autoimmune diseases.

Indexed as

Disease Models, AnimalAnimalsArthritis, RheumatoidAutoimmunityCartilageCartilage Oligomeric Matrix ProteinExtracellular Matrix ProteinsGlycoproteinsHumansMatrilin ProteinsPolychondritis, RelapsingRatsRats, Inbred LewCartilage Oligomeric Matrix ProteinExtracellular Matrix ProteinsGlycoproteinsMATN1 protein, humanMatrilin ProteinsTSP5 protein, human

Identifiers

PMID12223103
PMCPMC128937

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.