ArticleBritish journal of pharmacology2002
Endothelin-1-induced venous contraction is maintained in DOCA-salt hypertension; studies with receptor agonists.
Article in British journal of pharmacology, 2002. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 10 papers.
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10 citing papers in PubMed, 30 citations in OpenAlex.
- DOCA-salt hypertension impairs artery function in rat middle cerebral artery and parenchymal arterioles.Microcirculation (New York, N.Y. : 1994) · 2016Article
- Divergent signaling mechanisms for venous versus arterial contraction as revealed by endothelin-1.Journal of vascular surgery · 2015Article
- Reverse-mode Na+/Ca2+ exchange is an important mediator of venous contraction.Pharmacological research · 2012Article
- Vascular endothelin receptor type B: structure, function and dysregulation in vascular disease.Biochemical pharmacology · 2012Review
- Modulators of the vascular endothelin receptor in blood pressure regulation and hypertension.Current molecular pharmacology · 2011Review
- Endothelin (ET)-1 inhibits nicotinamide adenine dinucleotide phosphate oxidase activity in human abdominal aortic endothelial cells: a novel function of ETB1 receptors.Endocrinology · 2008Article
- A comparison of reactive oxygen species metabolism in the rat aorta and vena cava: focus on xanthine oxidase.American journal of physiology. Heart and circulatory physiology · 2008Article
- Pharmacological endothelin receptor interaction does not occur in veins from ET(B) receptor deficient rats.Vascular pharmacology · 2008Article
- The vascular endothelin system in hypertension--recent patents and discoveries.Recent patents on cardiovascular drug discovery · 2006Review
- Big ET-1 processing into vasoactive peptides in arteries and veins.Vascular pharmacologyArticle
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4 authors at 1 institution in 1 country.
Funding
Abstract
1. Deoxycorticosterone acetate (DOCA) salt hypertension is associated with an endothelin-1 (ET-1)-dependent increase in arterial resistance and mean circulatory filling pressure. Contraction of endothelium-intact arteries and veins from sham and DOCA-salt hypertensive rats to agonists of the ET(A) (ET-1((1-31))) and ET(B) receptor (sarafotoxin 6c; S6c) was investigated in tissue baths as was expression of mRNA for ET-1 and mRNA and protein for the ET(A) and ET(B) receptor. 2. ET-1((1-31)) contracted aorta and vena cava from sham rats with a 30 fold lower potency than ET-1. Contraction was not altered by the ET(B) receptor antagonist BQ788 (100 nM) but was abolished by the ET(A) receptor antagonist ABT-627 (30 nM). 3. In DOCA-salt thoracic aorta, maximum contraction to ET-1 and ET-1((1-31)) was reduced (36.6 +/- 6.3 and 13.3 +/- 4.4% of sham response, respectively); aorta did not contract to S6c. 4. In vena cava from DOCA-salt rats, contraction to ET-1 and ET-1((1-31)) was not reduced compared to sham contraction; vena cava from sham and DOCA-salt rats contracted to S6c with a similar potency. 5. Real time RT-PCR revealed that prepro ET-1 mRNA was increased 6.6 +/- 3.3 fold and 8.7 +/- 3.9 fold greater in DOCA-salt aorta and vena cava, respectively, compared to sham. Vena cava expressed a higher content of ET(A) and ET(B) receptor mRNA than aorta (P < 0.05), but no differences were observed between sham and DOCA-salt tissues. ET(A) and ET(B) receptor protein was identified in all tissues. Immunoreactive ET(A) receptor, observed as a 65, 30 and 28 kDa bands, was expressed 400% greater in DOCA-salt aorta compared to sham, but was not altered in vena cava. Immunoreactive ET(B) receptor, observed as 120, 45 and 30 kDa bands, tended to be higher in vena cava compared to aorta, but was not different in sham and DOCA-salt vena cava. 6. These results suggest that ET(A) receptor function is impaired in aorta but not vena cava of DOCA-salt rats. The ET(B) receptor was present in the aorta but, unlike in veins, does not mediate contraction directly. A sustained response to ET-1 in the venous circulation may contribute to the elevated blood pressure in the DOCA-salt model.
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