ArticleJournal of clinical microbiology2002
Prevalence of Helicobacter pylori in Sri Lanka as determined by PCR.
Article in Journal of clinical microbiology, 2002. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 8 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
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Who cites it
8 citing papers in PubMed.
- Comparison of gastric emptying of solid and semi-solid meals using real-time ultrasonography in healthy Sri Lankan adults.World journal of gastrointestinal pathophysiology · 2025Article
- Pepsinogen and Serum IgG Detection Is a Valuable Diagnostic Method forDiagnostics (Basel, Switzerland) · 2021Article
- Article
- Prevalence of Helicobacter pylori vacA, cagA, and iceA Genotypes in Cuban Patients with Upper Gastrointestinal Diseases.BioMed research international · 2015Article
- Presence of Helicobacter pylori in betel chewers and non betel chewers with and without oral cancers.BMC oral health · 2009Article
- Causal role of Helicobacter pylori infection in gastric cancer: an Asian enigma.World journal of gastroenterology · 2006Review
- Helicobacter pylori virulence factors in duodenal ulceration: A primary cause or a secondary infection causing chronicity.World journal of gastroenterology · 2006Article
- Prevalence of Helicobacter pylori vacA and cagA genotypes in Ethiopian dyspeptic patients.Journal of clinical microbiology · 2004Article
Corrections and comments
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Authors and funding
5 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Fifty-seven Sinhalese patients were investigated for the presence of Helicobacter pylori by PCR. A prevalence of 70.1%, with 47.5% positive for cagA, was demonstrated. The most common vacA allele was s1am1. There was no significant association between either the s1 allele or the cagA allele and severe gastroduodenal disease. There was an association between the s1 allele and the cagA locus.
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Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.