ReviewCurrent allergy and asthma reports2002
The genetics of otitis media.
Review in Current allergy and asthma reports, 2002. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 3 papers.
What it found
Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.
The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
The trial behind it
Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.
Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.
Who cites it
3 citing papers in PubMed.
- Innate Immunity: Orchestrating Inflammation and Resolution of Otitis Media.Current allergy and asthma reports · 2016Review
- TLR4-mediated induction of TLR2 signaling is critical in the pathogenesis and resolution of otitis media.Innate immunity · 2009Article
- Tympanometry assessment of 61 inbred strains of mice.Hearing research · 2007Article
Corrections and comments
PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.
Authors and funding
2 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Almost every child will suffer at least one episode of otitis media (OM). Therefore, it is not immediately obvious that there is a genetic predisposition to the development of the disease. However, evidence from a variety of studies has shown that there is a clear genetic component to susceptibility to OM. Methodologies for discovering these genes are described within this article. The identification of OM susceptibility genes allows the development of molecular diagnostic assays that will inform the clinician as to which child is at increased risk and warrants more aggressive intervention. Delineating the OM genes not only provides a deeper understanding of the pathophysiology of this common disease, but also of acute and chronic bacterial disease in general.
Indexed as
Identifiers
12044265What OpenQuestion holds
Registered trials
Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.