ArticleThe Journal of physiology2002
Rapidly and slowly activating components of delayed rectifier K(+) current in guinea-pig sino-atrial node pacemaker cells.
Article in The Journal of physiology, 2002. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 19 papers.
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Who cites it
19 citing papers in PubMed, 46 citations in OpenAlex.
- Characterization and functional role of rapid- and slow-activating delayed rectifier KPflugers Archiv : European journal of physiology · 2021Article
- Hypoglycaemia combined with mild hypokalaemia reduces the heart rate and causes abnormal pacemaker activity in a computational model of a human sinoatrial cell.Journal of the Royal Society, Interface · 2021Article
- Relative positioning of Kv11.1 (hERG) KScientific reports · 2018Article
- Functional characterization of Kv11.1 (hERG) potassium channels split in the voltage-sensing domain.Pflugers Archiv : European journal of physiology · 2018Article
- Heterogeneous functional expression of the sustained inward NaPflugers Archiv : European journal of physiology · 2018Article
- Ca2+/calmodulin potentiates I Ks in sinoatrial node cells by activating Ca2+/calmodulin-dependent protein kinase II.Pflugers Archiv : European journal of physiology · 2015Article
- Ionic mechanisms underlying the negative chronotropic action of propofol on sinoatrial node automaticity in guinea pig heart.British journal of pharmacology · 2015Article
- Inhibitory effects of sevoflurane on pacemaking activity of sinoatrial node cells in guinea-pig heart.British journal of pharmacology · 2012Article
- Postnatal developmental decline in IK1 in mouse ventricular myocytes isolated by the Langendorff perfusion method: comparison with the chunk method.Pflugers Archiv : European journal of physiology · 2012Article
- Autophagy is constitutively active in normal mouse sino-atrial nodal cells.Acta histochemica et cytochemica · 2011Article
- Voltage-dependent potassium currents in feline sino-atrial node myocytes.Pflugers Archiv : European journal of physiology · 2011Article
- A mathematical model of action potentials of mouse sinoatrial node cells with molecular bases.American journal of physiology. Heart and circulatory physiology · 2011Article
- Characterization of the rapidly activating delayed rectifier potassium current, I (Kr), in HL-1 mouse atrial myocytes.The Journal of membrane biology · 2010Article
- Multiple effects of 4-aminopyridine on feline and rabbit sinoatrial node myocytes and multicellular preparations.Pflugers Archiv : European journal of physiology · 2010Article
- Creating a cardiac pacemaker by gene therapy.Medical & biological engineering & computing · 2007Review
- Responses of the sustained inward current to autonomic agonists in guinea-pig sino-atrial node pacemaker cells.British journal of pharmacology · 2005Article
- Single-channel recordings of a rapid delayed rectifier current in adult mouse ventricular myocytes: basic properties and effects of divalent cations.The Journal of physiology · 2004Article
- Blocking action of chromanol 293B on the slow component of delayed rectifier K(+) current in guinea-pig sino-atrial node cells.British journal of pharmacology · 2002Article
- Potentiation of slow component of delayed rectifier K(+) current by cGMP via two distinct mechanisms: inhibition of phosphodiesterase 3 and activation of protein kinase G.British journal of pharmacology · 2002Article
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5 authors at 2 institutions in 1 country.
Funding
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Abstract
The components and properties of the delayed rectifier K(+) current (I(K)) in isolated guinea-pig sino-atrial (SA) node pacemaker cells were investigated using the whole-cell configuration of the patch-clamp technique. An envelope of tails test was conducted by applying depolarizing pulses from a holding potential of -50 mV to +30 mV for various durations ranging from 40 to 2000 ms. The ratio of the tail current amplitude elicited upon return to the holding potential to the magnitude of the time-dependent outward current activated during depolarizing steps was dependent on the pulse duration, while after exposure to the selective I(Kr) inhibitor E-4031 (5 microM) this current ratio became practically constant irrespective of the pulse duration. These observations are consistent with the presence of the E-4031-sensitive, rapidly activating and E-4031-resistant, slowly activating components of I(K) (I(Kr) and I(Ks), respectively) in guinea-pig SA node cells. The activation range for I(Kr), defined as the E-4031-sensitive current (half-maximal activation voltage (V(1/2)) of -26.2 mV) was much more negative than that for I(Ks), defined as the E-4031-resistant current (V(1/2) of +17.2 mV). I(Kr) exhibited a marked inward rectification at potentials positive to -50 mV, whereas I(Ks) showed only a slight rectification. In the current-clamp experiments, bath application of E-4031 (0.5 and 5 microM) initially slowed the repolarization at potentials negative to approximately -30 mV and produced a significant depolarization of the maximum diastolic potential, followed by the arrest of electrical activity, thus indicating that the late phase of the repolarization leading to the maximum diastolic potential at around -60 mV in spontaneous action potentials is primarily produced by I(Kr) in guinea-pig SA node cells. External application of the selective I(Ks) inhibitor 293B (30 microM) also delayed the repolarization process at potentials negative to about -20 mV and induced moderate depolarization of the maximum diastolic potential leading to the arrest of the spontaneous activity. These results provide evidence to suggest that both I(Kr) and I(Ks) are present and play crucial roles in the spontaneous electrical activity of guinea-pig SA node pacemaker cells.
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