Evidence map›Paper›PMID 11687010›Full record

SynthesisThe Cochrane database of systematic reviews2001

Routine intracranial pressure monitoring in acute coma.

R Forsyth, P Baxter, T Elliott

Abstract readSystematic Review
PubMed Publisher
In one paragraph

Synthesis in The Cochrane database of systematic reviews, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
5citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

5 citing papers in PubMed, 1 synthesis or guideline pooled it, 27 citations in OpenAlex.

  1. Routine intracranial pressure monitoring in acute coma.The Cochrane database of systematic reviews · 2015
    Pooled it
  2. Article
  3. Article
  4. Article
  5. BPSD following traumatic brain injury.Dementia & neuropsychologia
    Article
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

3 authors at 2 institutions in 1 country.

R ForsythDepartment of Child Health, University of Newcastle upon Tyne, Department of Child Health, Royal Victoria Infirmary, Newcastle upon Tyne, Tyne & Wear, UK, NE1 4LP. r.j.forsyth@newcastle.ac.uk
P Baxter
T Elliott
North Tyneside General Hospital · GBRoyal Victoria Infirmary · GB

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

backgroundStudies in traumatic encephalopathy first led to the insight that the damage seen was not just due to direct consequences of the primary injury. A significant, and potentially preventable, contribution to the overall morbidity arose from secondary hypoxic-ischaemic damage. Brain swelling accompanied by raised intracranial pressure (ICP) resulted in inadequate cerebral perfusion with well-oxygenated blood. Detection of raised ICP could be useful in alerting clinicians to the need to improve cerebral perfusion, with consequent reductions in brain injury.

objectivesTo determine whether routine ICP monitoring in all acute cases of severe coma reduces the risk of all-cause mortality or severe disability at final follow up. SEARCH STRATEGY: We searched MEDLINE, EMBASE, The Injuries Group's specialised register and the reference lists of all relevant articles and review articles. In addition we searched the Index of Scientific and Technical Proceedings using "intracranial pressure" as keyword. SELECTION CRITERIA: All randomised controlled studies of real-time ICP monitoring by invasive or semi-invasive means in acute coma (traumatic or non-traumatic aetiology) versus no ICP monitoring (ie clinical assessment of ICP ) DATA COLLECTION AND ANALYSIS: Primary outcome measures were all-cause mortality and severe disability at the end of the follow up period. MAIN

resultsNo studies meeting the selection criteria have been identified to date. REVIEWER'S

conclusionsThere are no data from randomised controlled trials that can clarify the role of ICP monitoring in acute coma.

Indexed as

Cerebrovascular CirculationAcute DiseaseBrain InjuriesComaHumansIntracranial HypertensionIntracranial PressureMonitoring, Physiologic

Identifiers

PMID11687010
OpenAlexW4248647425

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.