ArticleJournal of bacteriology2001
Distribution of the SsuDAT1I restriction-modification system among different serotypes of Streptococcus suis.
Article in Journal of bacteriology, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 5 papers.
What it found
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
5 citing papers in PubMed.
- A Locus Encoding Variable Defense Systems against Invading DNA Identified in Streptococcus suis.Genome biology and evolution · 2017Article
- Distribution of Type I Restriction-Modification Systems in Streptococcus suis: An Outlook.Pathogens (Basel, Switzerland) · 2016Article
- Conservation of genomic localization and sequence content of Sau3AI-like restriction-modification gene cassettes among Listeria monocytogenes epidemic clone I and selected strains of serotype 1/2a.Applied and environmental microbiology · 2010Article
- Different foreign genes incidentally integrated into the same locus of the Streptococcus suis genome.Journal of bacteriology · 2005Article
- Evidence for lateral transfer of the Suilysin gene region of Streptococcus suis.Journal of bacteriology · 2002Article
Corrections and comments
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Authors and funding
4 authors.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The SsuDAT1I restriction-modification (R-M) system, which contains two methyltransferases and two restriction endonucleases with recognition sequence 5'-GATC-3', was first found in a field isolate of Streptococcus suis serotype 2. Isoschizomers of the R-M system were found in the same locus between purH and purD in a field isolate of serotype 1/2 and the reference strains of serotypes 3, 7, 23, and 26 among 29 strains of different serotypes examined in this study. The R-M gene sequences in serotypes 1/2, 3, 7, and 23 were very similar to those of SsuDAT1I, whereas those in serotype 26 were less similar. These results indicate intraspecies recombination among them and genetic divergence through their evolution.
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