ArticleThe Journal of general physiology2001
Molecular architecture of the voltage-dependent Na channel: functional evidence for alpha helices in the pore.
Article in The Journal of general physiology, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.
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Who cites it
14 citing papers in PubMed, 36 citations in OpenAlex.
- Predicting Structural Details of the Sodium Channel Pore Basing on Animal Toxin Studies.Frontiers in pharmacology · 2018Review
- Analysis of inter-residue contacts reveals folding stabilizers in P-loops of potassium, sodium, and TRPV channels.European biophysics journal : EBJ · 2016Article
- Cardiac Na Channels: Structure to Function.Current topics in membranes · 2016Article
- Evolutionary history of a complex adaptation: tetrodotoxin resistance in salamanders.Evolution; international journal of organic evolution · 2015Article
- Constraint shapes convergence in tetrodotoxin-resistant sodium channels of snakes.Proceedings of the National Academy of Sciences of the United States of America · 2012Article
- Possible roles of exceptionally conserved residues around the selectivity filters of sodium and calcium channels.The Journal of biological chemistry · 2011Article
- Congenital insensitivity to pain: novel SCN9A missense and in-frame deletion mutations.Human mutation · 2010Article
- Models of the structure and gating mechanisms of the pore domain of the NaChBac ion channel.Biophysical journal · 2008Article
- Sodium channels: ionic model of slow inactivation and state-dependent drug binding.Biophysical journal · 2007Article
- Modelling insecticide-binding sites in the voltage-gated sodium channel.The Biochemical journal · 2006Article
- Modeling P-loops domain of sodium channel: homology with potassium channels and interaction with ligands.Biophysical journal · 2005Article
- Accessibility of mid-segment domain IV S6 residues of the voltage-gated Na+ channel to methanethiosulfonate reagents.The Journal of physiology · 2004Article
- Using the deadly mu-conotoxins as probes of voltage-gated sodium channels.Toxicon : official journal of the International Society on Toxinology · 2004Review
- Three ENU-induced neurological mutations in the pore loop of sodium channel Scn8a (Na(v)1.6) and a genetically linked retinal mutation, rd13.Mammalian genome : official journal of the International Mammalian Genome Society · 2004Article
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Authors and funding
5 authors at 2 institutions in 1 country.
Funding
Abstract
The permeation pathway of the Na channel is formed by asymmetric loops (P segments) contributed by each of the four domains of the protein. In contrast to the analogous region of K channels, previously we (Yamagishi, T., M. Janecki, E. Marban, and G. Tomaselli. 1997. Biophys. J. 73:195-204) have shown that the P segments do not span the selectivity region, that is, they are accessible only from the extracellular surface. The portion of the P-segment NH(2)-terminal to the selectivity region is referred to as SS1. To explore further the topology and functional role of the SS1 region, 40 amino acids NH(2)-terminal to the selectivity ring (10 in each of the P segments) of the rat skeletal muscle Na channel were substituted by cysteine and expressed in tsA-201 cells. Selected mutants in each domain could be blocked with high affinity by externally applied Cd(2)+ and were resistant to tetrodotoxin as compared with the wild-type channel. None of the externally applied sulfhydryl-specific methanethiosulfonate reagents modified the current through any of the mutant channels. Both R395C and R750C altered ionic selectivity, producing significant increases in K(+) and NH(4)(+) currents. The pattern of side chain accessibility is consistent with a pore helix like that observed in the crystal structure of the bacterial K channel, KcsA. Structure prediction of the Na channel using the program PHDhtm suggests an alpha helix in the SS1 region of each domain channel. We conclude that each of the P segments undergoes a hairpin turn in the permeation pathway, such that amino acids on both sides of the putative selectivity filter line the outer mouth of the pore. Evolutionary conservation of the pore helix motif from bacterial K channels to mammalian Na channels identifies this structure as a critical feature in the architecture of ion selective pores.
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