Evidence map›Paper›PMID 11459980›Full record

ReviewProceedings of the National Academy of Sciences of the United States of America2001

Homologous DNA recombination in vertebrate cells.

E Sonoda, M Takata, Y M Yamashita, C Morrison, S Takeda

Open access · greenAbstract readReview
In one paragraph

Review in Proceedings of the National Academy of Sciences of the United States of America, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 49 papers.

0numbers the graph read from it
0cells of the map it votes in
49citing papers in PubMed
7.1field-weighted citation impact, top 2% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

49 citing papers in PubMed, 148 citations in OpenAlex.

  1. Review
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  5. Review
  6. A pathway for mitotic chromosome formation.Science (New York, N.Y.) · 2018
    Article
  7. Article
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  9. Article
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  11. Targeting of Nrf2 induces DNA damage signaling and protects colonic epithelial cells from ionizing radiation.Proceedings of the National Academy of Sciences of the United States of America · 2012
    Article
  12. Regulatory networks integrating cell cycle control with DNA damage checkpoints and double-strand break repair.Philosophical transactions of the Royal Society of London. Series B, Biological sciences · 2011
    Review
  13. Article
  14. Article
  15. Article
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  17. Review
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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors at 2 institutions in 2 countries.

E SonodaDepartment of Radiation Genetics, Faculty of Medicine, Kyoto University, Yoshida Konoe, Sakyo-ku, Kyoto 606-8501, Japan.
M Takata
Y M Yamashita
C Morrison
S Takeda
University of Edinburgh · GBKawasaki Medical School · JP

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

The RAD52 epistasis group genes are involved in homologous DNA recombination, and their primary structures are conserved from yeast to humans. Although biochemical studies have suggested that the fundamental mechanism of homologous DNA recombination is conserved from yeast to mammals, recent studies of vertebrate cells deficient in genes of the RAD52 epistasis group reveal that the role of each protein is not necessarily the same as that of the corresponding yeast gene product. This review addresses the roles and mechanisms of homologous recombination-mediated repair with a special emphasis on differences between yeast and vertebrate cells.

Indexed as

DNA RepairRecombination, GeneticAllelesAnimalsCell SurvivalChickensDNA-Binding ProteinsDNA DamageFungal ProteinsHumansRad52 DNA Repair and Recombination ProteinSaccharomyces cerevisiaeSaccharomyces cerevisiae ProteinsVertebratesDNA-Binding ProteinsFungal ProteinsRad52 DNA Repair and Recombination ProteinRAD52 protein, humanRAD52 protein, S cerevisiaeSaccharomyces cerevisiae ProteinsX-ray repair cross complementing protein 3

Identifiers

PMID11459980
PMCPMC37448
OpenAlexW2110819297

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.