ArticleImmunology2001
Detection of anaphylatoxin receptors on CD83+ dendritic cells derived from human skin.
Article in Immunology, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 17 papers.
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Who cites it
17 citing papers in PubMed, 45 citations in OpenAlex.
- The complement system in human pregnancy and preeclampsia.Frontiers in immunology · 2025Review
- Complement C3 deficient mice show more severe imiquimod-induced psoriasiform dermatitis than wild-type mice regardless of the commensal microbiota.Experimental animals · 2024Article
- A bibliometric analysis: CaFrontiers in immunology · 2023Review
- Targeting the Complement Pathway as a Therapeutic Strategy in Lung Cancer.Frontiers in immunology · 2019Review
- Innate and adaptive immunologic functions of complement in the host response to Listeria monocytogenes infection.Immunobiology · 2016Review
- The receptor for the complement C3a anaphylatoxin (C3aR) provides host protection against Listeria monocytogenes-induced apoptosis.Journal of immunology (Baltimore, Md. : 1950) · 2014Article
- Role of C3a receptors, C5a receptors, and complement protein C6 deficiency in collagen antibody-induced arthritis in mice.Journal of immunology (Baltimore, Md. : 1950) · 2012Article
- Regulation of human mast cell and basophil function by anaphylatoxins C3a and C5a.Immunology letters · 2010Review
- Complement activation by CpG in a human whole blood loop system: mechanisms and immunomodulatory effects.Journal of immunology (Baltimore, Md. : 1950) · 2009Article
- C3a receptor deficiency accelerates the onset of renal injury in the MRL/lpr mouse.Molecular immunology · 2009Article
- The role of complement in danger sensing and transmission.Immunologic research · 2006Review
- Expression of complement protein C5a in a murine mammary cancer model: tumor regression by interference with the cell cycle.Cancer immunology, immunotherapy : CII · 2005Article
- New insights into the role of the complement pathway in allergy and asthma.Current allergy and asthma reports · 2005Review
- Anaphylatoxin C3a receptors in asthma.Respiratory research · 2005Review
- The anaphylatoxin C3a downregulates the Th2 response to epicutaneously introduced antigen.The Journal of clinical investigation · 2004Article
- Human monocyte-derived dendritic cells are chemoattracted to C3a after up-regulation of the C3a receptor with interferons.Immunology · 2004Article
- Up-regulation of C5a receptor expression and function on human monocyte derived dendritic cells by prostaglandin E2.Immunology · 2003Article
Corrections and comments
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Authors and funding
7 authors at 2 institutions in 1 country.
Funding
No grant is acknowledged in the PubMed record.
Abstract
Dendritic cells (DC) are recruited to sites of inflammation for the initiation of immune responses. As the anaphylatoxins C5a and C3a are important mediators of inflammation, we investigated the expression of their receptors (C3aR and C5aR) on human DC. DC were isolated from human skin or generated from purified blood monocytes and were identified by their expression of CD1a or CD83. Freshly isolated or cultured dermal CD1a+ and CD83+ DC bound anti-C5aR and anti-C3aR monoclonal antibodies (mAbs), as detected by flow cytometry. C5a induced calcium fluxes in dermal CD1a+ and CD83+ DC, which could be inhibited by C17/5, an anti-C5a mAb. C3a did not induce calcium fluxes in these cells. Anaphylatoxin receptor expression was down-regulated on dermal DC by adding tumour necrosis factor-alpha (TNF-alpha) to the culture medium. On CD1a+ CD83- cells generated from isolated blood monocytes by culture with 6.25 ng/ml of granulocyte-macrophage colony-stimulating factor (GM-CSF) and 125 U/ml of interleukin-4 (IL-4), expression of both C5aR and C3aR was observed. In these cells, both C5a and C3a induced calcium fluxes. After addition of TNF-alpha to the culture medium, the majority of the CD1a+ cells expressed CD83+. These cells - expressing a phenotype of 'mature DC' - down-regulated the expression of the anaphylatoxin receptors and lost their reactivity to the respective ligands. Our results demonstrate the expression of the anaphylatoxin receptors C5aR and C3aR on human skin-derived DC and blood-derived cells expressing the DC-associated membrane molecule, CD1a. Furthermore, the expression of anaphylatoxin receptors on CD83+ dermal DC is indicative of an intermediate stage of maturation of these cells, which was not observed on in vitro-differentiated CD83+ cells.
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