Evidence map›Paper›PMID 11315417›Full record

ReviewMedicine, health care, and philosophy2001

A new model for the origins of chronic disease.

D J Barker

Abstract readReview
PubMed Publisher
In one paragraph

Review in Medicine, health care, and philosophy, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 41 papers.

0numbers the graph read from it
0cells of the map it votes in
41citing papers in PubMed
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

41 citing papers in PubMed.

  1. Article
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  6. Review
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  14. Review
  15. Review
  16. Prenatal hypoxia in rats increased blood pressure and sympathetic drive of the adult offspring.Hypertension research : official journal of the Japanese Society of Hypertension · 2016
    Article
  17. Article
  18. Article
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  20. Review
4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

1 author.

D J BarkerMRC Environmental Epidemiology Unit, Southampton General Hospital, Southampton, SO16 6YD, UK.

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Living things are often plastic during their early development and are moulded by the environment. Many human fetuses have to adapt to a limited supply of nutrients, and in doing so they permanently change their physiology and metabolism. These programmed changes may be the origins of a number of diseases in later life, including coronary heart disease, stroke, diabetes and hypertension.

Indexed as

Chronic DiseaseModels, BiologicalCausalityCoronary DiseaseEmbryonic and Fetal DevelopmentFemaleGenetics, MedicalHumansLife ExpectancyMaleNutritional Physiological PhenomenaPlacental InsufficiencyPregnancy

Identifiers

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.