Evidence map›Paper›PMID 11230170›Full record

ArticleGenome research2001

A combinatorial partitioning method to identify multilocus genotypic partitions that predict quantitative trait variation.

M R Nelson, S L Kardia, R E Ferrell, C F Sing

Registry-linked trialAbstract readComparative Study
In one paragraph

Article in Genome research, 2001. The graph could read no effect estimate from its abstract, so it casts no vote on the map. It is linked to trial NCT00005209 (Genetic Epidemiology of Coronary Heart Disease), which is not on this map. Cited by 142 papers, 2 of them syntheses that pooled it.

0numbers the graph read from it
0cells of the map it votes in
142citing papers in PubMed, 2 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

NCT00005209 completednot on this map

Genetic Epidemiology of Coronary Heart Disease

TypeobservationalSponsorNational Heart, Lung, and Blood Institute (NHLBI)Ran1987 to 2007ConditionsCardiovascular Diseases, Heart Diseases, Coronary Disease
3 · Its place in the literature

Who cites it

142 citing papers in PubMed, 2 syntheses or guidelines pooled it.

  1. Pooled it
  2. GWAS of longevity in CHARGE consortium confirms APOE and FOXO3 candidacy.The journals of gerontology. Series A, Biological sciences and medical sciences · 2015
    Pooled it
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82 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

4 authors.

M R NelsonDepartment of Human Genetics, University of Michigan, Ann Arbor, Michigan 48109-0618, USA.
S L Kardia
R E Ferrell
C F Sing

Funding

GENETIC EPIDEMIOLOGY OF CORONARY HEART DISEASER01HL039107 · NHLBI · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI SING, CHARLES F · 1987 to 2005
$5.8M
MODELING DNA DIVERSITY IN CARDIOVASCULAR HEALTH/DISEASER01HL058240 · NHLBI · UNIVERSITY OF MICHIGAN AT ANN ARBOR · PI SING, CHARLES F · 1997 to 2001
$1.2M
NHLBI NIH HHS HL39107NHLBI NIH HHS HL58240NHLBI NIH HHS R01 HL039107
6 · The paper itself

Abstract

Recent advances in genome research have accelerated the process of locating candidate genes and the variable sites within them and have simplified the task of genotype measurement. The development of statistical and computational strategies to utilize information on hundreds -- soon thousands -- of variable loci to investigate the relationships between genome variation and phenotypic variation has not kept pace, particularly for quantitative traits that do not follow simple Mendelian patterns of inheritance. We present here the combinatorial partitioning method (CPM) that examines multiple genes, each containing multiple variable loci, to identify partitions of multilocus genotypes that predict interindividual variation in quantitative trait levels. We illustrate this method with an application to plasma triglyceride levels collected on 188 males, ages 20--60 yr, ascertained without regard to health status, from Rochester, Minnesota. Genotype information included measurements at 18 diallelic loci in six coronary heart disease--candidate susceptibility gene regions: APOA1--C3--A4, APOB, APOE, LDLR, LPL, and PON1. To illustrate the CPM, we evaluated all possible partitions of two-locus genotypes into two to nine partitions (approximately 10(6) evaluations). We found that many combinations of loci are involved in sets of genotypic partitions that predict triglyceride variability and that the most predictive sets show nonadditivity. These results suggest that traditional methods of building multilocus models that rely on statistically significant marginal, single-locus effects, may fail to identify combinations of loci that best predict trait variability. The CPM offers a strategy for exploring the high-dimensional genotype state space so as to predict the quantitative trait variation in the population at large that does not require the conditioning of the analysis on a prespecified genetic model.

Indexed as

Genetic VariationMultigene FamilyQuantitative Trait, HeritableAdultCombinatorial Chemistry TechniquesComputational BiologyGenetic MarkersGenotypeHumansMaleMiddle AgedModels, GeneticPhenotypeReproducibility of ResultsGenetic Markers

Identifiers

PMID11230170
PMCPMC311041

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Registered trials

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.