The case for the involvement of the Upf3p in programmed -1 ribosomal frameshifting.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

4 citing papers in PubMed.

1. Evidence against a direct role for the Upf proteins in frameshifting or nonsense codon readthrough.RNA (New York, N.Y.) · 2004
   Article
2. An in vivo dual-luciferase assay system for studying translational recoding in the yeast Saccharomyces cerevisiae.RNA (New York, N.Y.) · 2003
   Article
3. A -1 ribosomal frameshift element that requires base pairing across four kilobases suggests a mechanism of regulating ribosome and replicase traffic on a viral RNA.Proceedings of the National Academy of Sciences of the United States of America · 2002
   Article
4. Ribosomal protein L5 helps anchor peptidyl-tRNA to the P-site in Saccharomyces cerevisiae.RNA (New York, N.Y.) · 2001
   Article

Corrections and comments

• Commented on by
  The case against the involvement of the NMD proteins in programmed frameshifting.2000
• Comment on
  Nonsense-mediated decay mutants do not affect programmed -1 frameshifting.2000

Authors and funding

4 authors.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as