Evidence map›Paper›PMID 11138614›Full record

ReviewJournal of clinical laboratory analysis2000

Progress in the use of biochemical and biological markers for evaluation of rheumatoid arthritis.

R M Nakamura

Open access · bronzeAbstract readReview
In one paragraph

Review in Journal of clinical laboratory analysis, 2000. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 14 papers.

0numbers the graph read from it
0cells of the map it votes in
14citing papers in PubMed
1.0field-weighted citation impact, top 22% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

14 citing papers in PubMed, 72 citations in OpenAlex.

  1. Review
  2. Article
  3. Article
  4. Article
  5. Article
  6. Article
  7. Article
  8. Autoimmunity against fibrinogen mediates inflammatory arthritis in mice.Journal of immunology (Baltimore, Md. : 1950) · 2010
    Article
  9. Review
  10. Article
  11. Review
  12. Article
  13. Article
  14. Review
4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

1 author at 1 institution in 1 country.

R M NakamuraDepartment of Pathology, Scripps Clinic, La Jolla, California 92037, USA.
Scripps Clinic · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Rheumatoid arthritis (RA) is a chronic systemic inflammatory autoimmune disorder which is predominant in females. The exact etiology remains undefined. Recently, a large number of biochemical and biologic markers, which are useful in the diagnosis, prognosis, and monitoring therapy of RA, have been reported. The new markers include genetic markers, filaggrin, citrulline containing peptides, A2/RA33, cytokines, joint and collagen breakdown products, and bone turnover markers. No laboratory tests in and of themselves are diagnostic of RA. The new markers have been employed in monitoring RA patients during treatment and following the course of the disease. With the development of innovative therapies for RA, many of the biochemical and biologic markers will be useful.

Indexed as

Acute-Phase ProteinsAntibodies, AntinuclearArthritis, RheumatoidBiomarkersBone RemodelingCollagenCytokinesFilaggrin ProteinsHumansIntermediate Filament ProteinsJointsPrognosisRheumatoid FactorAcute-Phase ProteinsAntibodies, AntinuclearBiomarkersCollagenCytokinesFilaggrin ProteinsFLG protein, humanIntermediate Filament ProteinsRheumatoid Factor

Identifiers

PMID11138614
PMCPMC6808153
OpenAlexW2101772859

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.