Evidence map›Paper›PMID 10970394›Full record

ArticleJournal of clinical microbiology2000

Distribution of rotavirus VP7 serotypes and VP4 genotypes circulating in Sousse, Tunisia, from 1995 to 1999: emergence of natural human reassortants.

A Trabelsi, I Peenze, C Pager, M Jeddi, D Steele

Abstract read
In one paragraph

Article in Journal of clinical microbiology, 2000. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 13 papers, 1 of them a synthesis that pooled it.

0numbers the graph read from it
0cells of the map it votes in
13citing papers in PubMed, 1 pooled it
–field-weighted citation impact
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

13 citing papers in PubMed, 1 synthesis or guideline pooled it.

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4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

5 authors.

A TrabelsiLaboratoire des Microbiologie-Immunologie, Hopital Universitaire, Farhat Hached, Sousse, Tunisia.
I Peenze
C Pager
M Jeddi
D Steele

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Rotavirus strains circulating in Sousse, Tunisia, between 1995 and 1999 were characterized antigenically by monoclonal antibodies to the VP6 subgroup and the VP7 serotype. The VP4 genotype was determined by reverse transcription-PCR, as were the strains with untyped VP7. Only 17% of 375 children were shedding rotavirus as determined by latex agglutination assay. Most rotavirus strains were G1P[8] (50%), followed by G4P[8] and G4P[6]. Reassortant G1P[4] strains emerged in Sousse during the 1998-1999 season.

Indexed as

Antigens, ViralCapsid ProteinsRecombination, GeneticCapsidChildChild, PreschoolElectrophoresis, Polyacrylamide GelEnzyme-Linked Immunosorbent AssayGenotypeHumansInfantReassortant VirusesRotavirusRotavirus InfectionsSensitivity and SpecificitySerotypingAntigens, ViralCapsid ProteinsVP4 protein, RotavirusVP6 protein, RotavirusVP7 protein, Rotavirus

Identifiers

PMID10970394
PMCPMC87397

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.