ArticleMolecular psychiatry2000
Analysis of autism susceptibility gene loci on chromosomes 1p, 4p, 6q, 7q, 13q, 15q, 16p, 17q, 19q and 22q in Finnish multiplex families.
Article in Molecular psychiatry, 2000. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 20 papers.
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The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.
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Who cites it
20 citing papers in PubMed, 54 citations in OpenAlex.
- Article
- AMPD1 functional variants associated with autism in Han Chinese population.European archives of psychiatry and clinical neuroscience · 2015Article
- Common genetic variants on 1p13.2 associate with risk of autism.Molecular psychiatry · 2014Article
- Triggers for autism: genetic and environmental factors.Journal of central nervous system disease · 2012Article
- Gene expression profiling differentiates autism case-controls and phenotypic variants of autism spectrum disorders: evidence for circadian rhythm dysfunction in severe autism.Autism research : official journal of the International Society for Autism Research · 2009Article
- Sequencing and analyzing the t(1;7) reciprocal translocation breakpoints associated with a case of childhood-onset schizophrenia/autistic disorder.Journal of autism and developmental disorders · 2008Article
- Early pharmacological treatment of autism: a rationale for developmental treatment.Biological psychiatry · 2007Review
- Analysis of case-parent trios at a locus with a deletion allele: association of GSTM1 with autism.BMC genetics · 2006Article
- Identification of significant association and gene-gene interaction of GABA receptor subunit genes in autism.American journal of human genetics · 2005Article
- Allelic heterogeneity at the serotonin transporter locus (SLC6A4) confers susceptibility to autism and rigid-compulsive behaviors.American journal of human genetics · 2005Article
- Genetic advances in autism hinge on the method of measuring symptoms.Current psychiatry reports · 2005Review
- A genetic study of autism in Costa Rica: multiple variables affecting IQ scores observed in a preliminary sample of autistic cases.BMC psychiatry · 2005Article
- Cytogenetic abnormalities and fragile-X syndrome in Autism Spectrum Disorder.BMC medical genetics · 2005Article
- Genome-wide and Ordered-Subset linkage analyses provide support for autism loci on 17q and 19p with evidence of phenotypic and interlocus genetic correlates.BMC medical genetics · 2005Article
- Linkage analysis for autism in a subset families with obsessive-compulsive behaviors: evidence for an autism susceptibility gene on chromosome 1 and further support for susceptibility genes on chromosome 6 and 19.Molecular psychiatry · 2004Article
- Molecular genetics of autism spectrum disorders.Journal of human genetics · 2003Review
- On the twin risk in autism.American journal of human genetics · 2002Article
- A genomewide screen for autism-spectrum disorders: evidence for a major susceptibility locus on chromosome 3q25-27.American journal of human genetics · 2002Article
- Excess of twins among affected sibling pairs with autism: implications for the etiology of autism.American journal of human genetics · 2001Article
- Evidence for a susceptibility gene for autism on chromosome 2 and for genetic heterogeneity.American journal of human genetics · 2001Article
Corrections and comments
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Authors and funding
6 authors at 2 institutions in 2 countries.
Funding
No grant is acknowledged in the PubMed record.
Abstract
The role of genetic factors in the etiology of the autistic spectrum of disorders has clearly been demonstrated. Ten chromosomal regions, on chromosomes 1p, 4p, 6q, 7q, 13q, 15q, 16p, 17q, 19q and 22q have potentially been linked to autism.1-8 We have analyzed these chromosomal regions in a total of 17 multiplex families with autism originating from the isolated Finnish population by pairwise linkage analysis and sib-pair analysis. Mild evidence for putative contribution was found only with the 1p chromosomal region in the susceptibility to autism. Our data suggest that additional gene loci exist for autism which will be detectable in and even restricted to the isolated Finnish population.
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