Evidence map›Paper›PMID 10819501›Full record

ReviewJournal of mammary gland biology and neoplasia1998

Mammary gland growth and development from the postnatal period to postmenopause: ovarian steroid receptor ontogeny and regulation in the mouse.

J L Fendrick, A M Raafat, S Z Haslam

Abstract readReview
PubMed Publisher
In one paragraph

Review in Journal of mammary gland biology and neoplasia, 1998. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 54 papers.

0numbers the graph read from it
0cells of the map it votes in
54citing papers in PubMed
6.0field-weighted citation impact, top 3% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

54 citing papers in PubMed, 143 citations in OpenAlex.

  1. Article
  2. Review
  3. Ovarian Accumulation of Nanoemulsions: Impact of Mice Age and Particle Size.International journal of molecular sciences · 2021
    Article
  4. Expression of Grainyhead-like 2 in the Process of Ductal Development of Mouse Mammary Gland.The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society · 2021
    Article
  5. Development and Pathology of the Equine Mammary Gland.Journal of mammary gland biology and neoplasia · 2021
    Review
  6. Article
  7. Article
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  11. Review
  12. Article
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4 · The record

Corrections and comments

5 · Who and what money

Authors and funding

3 authors at 1 institution in 1 country.

J L FendrickDepartment of Physiology, Michigan State University, East Lansing 48824-1101, USA.
A M Raafat
S Z Haslam
Michigan State University · US

Funding

STROMA AND MAMMARY GLAND CELL PROLIFERATIONR01CA040104 · NCI · MICHIGAN STATE UNIVERSITY · PI HASLAM, SANDRA Z · 1985 to 2007
$1.6M
HORMONAL RESPONSIVENESS OF POSTMENOPAUSAL MAMMARY GLANDR01AG013059 · NIA · MICHIGAN STATE UNIVERSITY · PI HASLAM, SANDRA Z · 1994 to 1997
–
NCI NIH HHS CA40104NIA NIH HHS AG13059
6 · The paper itself

Abstract

Ovarian steroid hormones play a critical role in regulating mammary gland growth and development. The mammary gland sequentially acquires and cyclically exhibits proliferative responses to estrogen and/or progesterone from birth to postmenopause. The focus of this review is to present our current understanding of estrogen and progesterone receptor distribution in epithelial and stromal cells and their functions in relation to mammary gland development. Insights gained from the study of the normal mammary gland are relevant to our understanding of the conditions which may predispose women to the development of breast cancer as well as to alterations in hormonal regulation that occur in breast cancer.

Indexed as

AnimalsBreastEstrogensFemaleHumansMammary Glands, AnimalMiceOvaryPostmenopauseProgesteroneReceptors, EstrogenReceptors, ProgesteroneEstrogensProgesteroneReceptors, EstrogenReceptors, Progesterone

Identifiers

PMID10819501
OpenAlexW2412849637

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.