Meeting Koch's postulates for calcium signaling in cardiac hypertrophy.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

14 citing papers in PubMed.

1. Effects of Long-Term Administration of Bovine Bone Gelatin Peptides on Myocardial Hypertrophy in Spontaneously Hypertensive Rats.Nutrients · 2023
   Article
2. BET bromodomain proteins regulate transcriptional reprogramming in genetic dilated cardiomyopathy.JCI insight · 2020
   Article
3. Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarction.Circulation research · 2014
   Article
4. Differential metal content and gene expression in rat left ventricular hypertrophy due to hypertension and hyperactivity.Journal of trace elements in medicine and biology : organ of the Society for Minerals and Trace Elements (GMS) · 2014
   Article
5. Calcineurin mediates bladder wall remodeling secondary to partial outlet obstruction.American journal of physiology. Renal physiology · 2011
   Article
6. Endoplasmic reticulum-mitochondria crosstalk in NIX-mediated murine cell death.The Journal of clinical investigation · 2009
   Article
7. The Ca2+ ATPase of cardiac sarcoplasmic reticulum: Physiological role and relevance to diseases.Biochemical and biophysical research communications · 2008
   Review
8. CaMKII, an emerging molecular driver for calcium homeostasis, arrhythmias, and cardiac dysfunction.Journal of molecular medicine (Berlin, Germany) · 2007
   Review
9. Changes of expression of stretch-activated potassium channel TREK-1 mRNA and protein in hypertrophic myocardium.Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban · 2006
   Article
10. Activation of nuclear factor-kappaB is necessary for myotrophin-induced cardiac hypertrophy.The Journal of cell biology · 2002
    Article
11. Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo.Proceedings of the National Academy of Sciences of the United States of America · 2002
    Article
12. Activation of NF-kappa B is required for hypertrophic growth of primary rat neonatal ventricular cardiomyocytes.Proceedings of the National Academy of Sciences of the United States of America · 2001
    Article
13. An abnormal Ca(2+) response in mutant sarcomere protein-mediated familial hypertrophic cardiomyopathy.The Journal of clinical investigation · 2000
    Article
14. Flow in the early embryonic human heart: a numerical study.Pediatric cardiology
    Article

Corrections and comments

• Commented on by
  CaM kinase signaling induces cardiac hypertrophy and activates the MEF2 transcription factor in vivo.2000
• Commented on by
  Re-evaluating sarcoplasmic reticulum function in heart failure.2000

Authors and funding

1 author.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

Indexed as