ArticleThe American journal of pathology2000
p53 mutations and expression in breast carcinoma in situ.
Article in The American journal of pathology, 2000. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 16 papers, 1 of them a synthesis that pooled it.
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Who cites it
16 citing papers in PubMed, 1 synthesis or guideline pooled it, 48 citations in OpenAlex.
- The association between phosphatase and tensin homolog hypermethylation and patients with breast cancer, a meta-analysis and literature review.Scientific reports · 2016Pooled it
- Elucidating cooperative genetic events in DCIS progression in mutantProceedings of the National Academy of Sciences of the United States of America · 2026Article
- Is loss of p53 a driver of ductal carcinoma in situ progression?British journal of cancer · 2022Review
- Determination of expression profile of p53 gene in different grades of breast cancer tissues by real time PCR.African health sciences · 2020Article
- Intratumoral Heterogeneity in Ductal Carcinoma In Situ: Chaos and Consequence.Journal of mammary gland biology and neoplasia · 2018Review
- Mortality risk of black women and white women with invasive breast cancer by hormone receptors, HER2, and p53 status.BMC cancer · 2013Article
- p53 mutations in classic and pleomorphic invasive lobular carcinoma of the breast.Cellular oncology (Dordrecht, Netherlands) · 2012Article
- Pathological characteristics of BRCA-associated breast cancers in Hispanics.Breast cancer research and treatment · 2011Article
- New aspects of mitochondrial Uncoupling Proteins (UCPs) and their roles in tumorigenesis.International journal of molecular sciences · 2011Review
- Frequent aberrant DNA methylation of ABCB1, FOXC1, PPP2R2B and PTEN in ductal carcinoma in situ and early invasive breast cancer.Breast cancer research : BCR · 2010Article
- Mutations in p53, p53 protein overexpression and breast cancer survival.Journal of cellular and molecular medicine · 2009Article
- Detection of HER-2/neu, c-myc amplification and p53 inactivation by FISH in Egyptian patients with breast cancer.German medical science : GMS e-journal · 2009Article
- Serum organochlorines and breast cancer: a case-control study among African-American women.Cancer causes & control : CCC · 2007Article
- High-throughput genomic technology in research and clinical management of breast cancer. Molecular signatures of progression from benign epithelium to metastatic breast cancer.Breast cancer research : BCR · 2006Review
- Mammary epithelial cell transformation: insights from cell culture and mouse models.Breast cancer research : BCR · 2005Review
- The p53 tumor suppressor gene in ductal carcinoma in situ of the breast.The American journal of pathology · 2000Review
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3 authors at 1 institution in 1 country.
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Abstract
The p53 tumor suppressor gene is altered in approximately half of human cancers. Although p53 mutations are common in invasive breast carcinoma, few have been identified in breast carcinoma in situ (intraductal breast carcinomas). Most studies of p53 in breast carcinoma in situ are immunohistochemical studies of p53 staining in paraffin-embedded tissue sections. Few studies have isolated the tumor cells and subjected them to DNA sequence analysis. The current study was undertaken to characterize p53 in a cohort of breast carcinoma in situ cases, both with and without invasive disease. Fifty-eight frozen breast biopsy samples were used for these investigations. Twenty-seven cases had only ductal carcinoma in situ (CIS) and 31 cases had evidence of both invasive and in situ carcinoma. DNA sequence alterations in exons 2 through 11 of p53 were screened by the single-strand conformational polymorphism technique. Exons with altered mobility were sequenced. Among breast CIS cases without invasive disease, 22% had p53 mutations and 7% had DNA sequence alterations of unknown significance. Analysis of breast CIS with concurrent invasive disease demonstrated p53 mutations in 19% of cases and one (3%) DNA alteration of unknown significance. Each carcinoma having a p53 mutation in the breast CIS component had the identical mutation in the invasive component of the same tumor indicating a clonal relationship between the two tumor components. p53 protein overexpression was identified in 22% of pure intraductal breast carcinomas and in 35% of breast CIS with invasive disease. Comparison of immunostaining and DNA sequence alterations showed a significant association between overexpression and mutations (P = 0. 0037) in cases of CIS without invasion, and similarly between overexpression and mutations in cases of CIS with invasion (P = 0. 007). p53 mutations and p53 overexpression were relatively common in intraductal breast carcinomas but were not observed in adjacent normal breast lobules or ducts in 9 cases available for DNA analysis. The frequency of p53 alterations when comparing breast CIS with and without an invasive component indicated that p53 mutations usually occur before invasion during the progression of breast cancer, as is observed for a number of other adult solid tumors.
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