The development of B cells in the bone marrow is controlled by the balance between cell-autonomous mechanisms and signals from the microenvironment.

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

Who cites it

25 citing papers in PubMed.

1. Serum-free differentiation platform for the generation of B lymphocytes and natural killer cells from human CD34+ cord blood progenitors.Scientific reports · 2025
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2. Inferring gene regulatory networks by hypergraph generative model.Cell reports methods · 2025
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3. How the bone microenvironment shapes the pre-metastatic niche and metastasis.Nature cancer · 2024
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4. The RNA-binding protein hnRNP F is required for the germinal center B cell response.Nature communications · 2023
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5. Autophagy and Its Lineage-Specific Roles in the Hematopoietic System.Oxidative medicine and cellular longevity · 2023
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6. Comprehensive phenotyping of human peripheral blood B lymphocytes in healthy conditions.Cytometry. Part A : the journal of the International Society for Analytical Cytology · 2022
   Review
7. Deletion ofFrontiers in immunology · 2022
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8. Cutting Edge: Myosin 18A Is a Novel Checkpoint Regulator in B Cell Differentiation and Antibody-Mediated Immunity.Journal of immunology (Baltimore, Md. : 1950) · 2021
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9. Stable colony-stimulating factor 1 fusion protein treatment increases hematopoietic stem cell pool and enhances their mobilisation in mice.Journal of hematology & oncology · 2021
   Article
10. Strategies for enhancing adoptive T-cell immunotherapy against solid tumors using engineered cytokine signaling and other modalities.Expert opinion on biological therapy · 2018
    Review
11. Immunosuppression in Experimental Chagas Disease Is Mediated by an Alteration of Bone Marrow Stromal Cell Function During the Acute Phase of Infection.Frontiers in immunology · 2018
    Article
12. Thymic B cell development is controlled by the B potential of progenitors via both hematopoietic-intrinsic and thymic microenvironment-intrinsic regulatory mechanisms.PloS one · 2018
    Article
13. The Complex Interplay between DNA Injury and Repair in Enzymatically Induced Mutagenesis and DNA Damage in B Lymphocytes.International journal of molecular sciences · 2017
    Review
14. Fra-2 regulates B cell development by enhancing IRF4 and Foxo1 transcription.The Journal of experimental medicine · 2017
    Article
15. Enhanced hematopoietic stem cell function mediates immune regeneration following sex steroid blockade.Stem cell reports · 2015
    Article
16. Arsenite selectively inhibits mouse bone marrow lymphoid progenitor cell development in vivo and in vitro and suppresses humoral immunity in vivo.PloS one · 2014
    Article
17. n-3 PUFAs enhance the frequency of murine B-cell subsets and restore the impairment of antibody production to a T-independent antigen in obesity.Journal of lipid research · 2013
    Article
18. B-Cell Response during Protozoan Parasite Infections.Journal of parasitology research · 2012
    Article
19. Distinct roles of IL-7 and stem cell factor in the OP9-DL1 T-cell differentiation culture system.Experimental hematology · 2006
    Article
20. 3BP2 deficiency impairs the response of B cells, but not T cells, to antigen receptor ligation.Molecular and cellular biology · 2006
    Article

Corrections and comments

• Comment on
  Precursor B cell receptor-dependent B cell proliferation and differentiation does not require the bone marrow or fetal liver environment.2000

Authors and funding

1 author.

Funding

No grant is acknowledged in the PubMed record.

Abstract

PubMed holds no abstract for this paper.

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