Evidence map›Paper›PMID 10586891›Full record

ReviewNature1999

Induction and regulation of the IgE response.

D B Corry, F Kheradmand

Abstract readReview
PubMed Publisher
In one paragraph

Review in Nature, 1999. The graph could read no effect estimate from its abstract, so it casts no vote on the map. Cited by 68 papers.

0numbers the graph read from it
0cells of the map it votes in
68citing papers in PubMed
13.5field-weighted citation impact, top 1% of its field
1 · What the graph read from it

What it found

Each row is one number read from the abstract, on the scale the paper reported it, with its interval. Left of the dashed line favours the treatment, right favours the comparator. Under each row is the sentence it came from. New to these charts? A ten-minute tutorial.

The abstract states no effect estimate the extractor could read, or names no intervention and outcome on the map, so this paper lights no cell and moves no belief. It is still indexed, cited and linked below.

2 · The registry

The trial behind it

Trials whose registry record cites this paper, or whose number appears in the abstract. A trial that started after this paper was published is citing it as background, not reporting it.

Neither the registry nor the abstract names a trial number. If this is a trial report, that itself is worth knowing.

3 · Its place in the literature

Who cites it

68 citing papers in PubMed, 323 citations in OpenAlex.

  1. Trial
  2. Article
  3. Article
  4. Article
  5. Cloaking antibodies are prevalent inFrontiers in cellular and infection microbiology · 2024
    Article
  6. Article
  7. Article
  8. Article
  9. Review
  10. Article
  11. Article
  12. Article
  13. Review
  14. Review
  15. Article
  16. The Role of CD4International journal of molecular sciences · 2021
    Review
  17. Article
  18. Article
  19. Long-Term Successful Treatment of Indolent Systemic Mastocytosis With Omalizumab.Federal practitioner : for the health care professionals of the VA, DoD, and PHS · 2021
    Article
  20. Observational

8 more citing papers are in PubMed but not listed here.

4 · The record

Corrections and comments

PubMed lists nothing against this paper. Absence here is not a guarantee, only a check that was made.

5 · Who and what money

Authors and funding

2 authors at 1 institution in 1 country.

D B CorryDepartment of Medicine, Baylor College of Medicine, Houston, Texas 77030, USA.
F Kheradmand
Baylor College of Medicine · US

Funding

No grant is acknowledged in the PubMed record.

6 · The paper itself

Abstract

Immunoglobulin E (IgE) is believed to be one of the major mediators of immediate hypersensitivity reactions that underlie atopic conditions such as urticaria, seasonal allergy, asthma and anaphylaxis. Factors that control IgE production are therefore essential to the pathogenesis of these important afflictions. But a complete understanding of this topic is lacking, while new data have raised questions regarding the precise role of IgE in atopic disease. Evolving concepts of IgE production and elimination are likely to clarify the importance of IgE in health and disease.

Indexed as

AllergensAnimalsForecastingHumansImmunoglobulin EInterleukin-13Interleukin-4Receptors, IgERespiratory HypersensitivitySignal TransductionT-Lymphocytes, Helper-InducerAllergensImmunoglobulin EInterleukin-13Interleukin-4Receptors, IgE

Identifiers

PMID10586891
OpenAlexW1993980743

What OpenQuestion holds

Textmetadata
Read underepoch 390

Registered trials

None linked

Read under generation 80e0d062 · epoch 390. Bibliography from PubMed, PubMed Central and OpenAlex; grants from NIH RePORTER; trial links from ClinicalTrials.gov; estimates, votes and beliefs from the OpenQuestion graph.